Results 231 to 240 of about 56,650 (275)
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Scandinavian Journal of Gastroenterology, 1987
The two final phases in the haemostatic process, plasma coagulation with the formation of a fibrin clot, and fibrinolysis leading to the dissolution of fibrin clots, are reviewed. Coagulation may be initiated either by reactions occurring between components of the blood alone, the intrinsic pathway, or by reactions which also involve tissue components,
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The two final phases in the haemostatic process, plasma coagulation with the formation of a fibrin clot, and fibrinolysis leading to the dissolution of fibrin clots, are reviewed. Coagulation may be initiated either by reactions occurring between components of the blood alone, the intrinsic pathway, or by reactions which also involve tissue components,
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Scandinavian Journal of Haematology, 1969
A clinical series of 40 patients was observed for 5 months, in the course of which every patient took 1.5 g. metformin daily for 3 months. No increase in fibrinolytic activity was demonstrable by euglobulin lysis test during the course of treatment. Nor did the fibrinogen and cholesterol values change. Blood glucose decreased.
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A clinical series of 40 patients was observed for 5 months, in the course of which every patient took 1.5 g. metformin daily for 3 months. No increase in fibrinolytic activity was demonstrable by euglobulin lysis test during the course of treatment. Nor did the fibrinogen and cholesterol values change. Blood glucose decreased.
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British Journal of Haematology
Fibrinolysis is the process by which fibrin, the principal proteinaceous component of blood clots, is enzymatically degraded. A key event in this process is the formation of the tissue plasminogen activator (tPA)–plasminogen complex on partially degraded fibrin, which leads to the conversion of plasminogen to plasmin, which then proteolytically digests
Anton Ilich, Nigel S. Key
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Fibrinolysis is the process by which fibrin, the principal proteinaceous component of blood clots, is enzymatically degraded. A key event in this process is the formation of the tissue plasminogen activator (tPA)–plasminogen complex on partially degraded fibrin, which leads to the conversion of plasminogen to plasmin, which then proteolytically digests
Anton Ilich, Nigel S. Key
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Nature, 1953
THE plasma of normal people has been shown to contain a labile, active fibrinolysin1,2,3. Since this enzyme is not necessarily plasmin and in some ways behaves differently from plasmin, it has been tentatively termed ‘plasma active fibrinolysin’2. The enzyme disappears rapidly from fluid blood and plasma kept at room temperature; but if blood is cooled
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THE plasma of normal people has been shown to contain a labile, active fibrinolysin1,2,3. Since this enzyme is not necessarily plasmin and in some ways behaves differently from plasmin, it has been tentatively termed ‘plasma active fibrinolysin’2. The enzyme disappears rapidly from fluid blood and plasma kept at room temperature; but if blood is cooled
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Pathophysiology of fibrinolysis
Journal of Internal Medicine, 1994P J, Declerck +3 more
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Fibrinolysis and Atherosclerosis
Seminars in Thrombosis and Hemostasis, 1988D, Collen, I, Juhan-Vague
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The Journal of Trauma: Injury, Infection, and Critical Care, 1969
B, Borowiecki, A A, Sharp
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B, Borowiecki, A A, Sharp
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