Results 291 to 300 of about 127,027 (324)
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Progress in Cardiovascular Diseases, 1979
NVESTIGATIONS during the past 3 decades have firmly established that the fibrinolytic system is an integral part of the hemostatic mechanism and that its derangement may contribute to the pathogenesis of thrombosis’” (this has been extensively reviewed in this symposium and elsewhere).
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NVESTIGATIONS during the past 3 decades have firmly established that the fibrinolytic system is an integral part of the hemostatic mechanism and that its derangement may contribute to the pathogenesis of thrombosis’” (this has been extensively reviewed in this symposium and elsewhere).
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Seminars in Thrombosis and Hemostasis, 1990
Although Trousseau 1 was the earliest observer who noted the association between cancer and thrombosis, it was Billroth 2 who first described the presence of tumor cells within a thrombus 2 .
Harold N. Keer, Hau C. Kwaan
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Although Trousseau 1 was the earliest observer who noted the association between cancer and thrombosis, it was Billroth 2 who first described the presence of tumor cells within a thrombus 2 .
Harold N. Keer, Hau C. Kwaan
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Thrombosis and Haemostasis, 1977
SummaryBlood platelets play a crucial role in the response of blood to vascular injury through adherence to exposed subendothelial vessel wall structures, the subsequent release of substances which trigger platelet aggregation, and release-associated changes in platelet surface properties which facilitate the interaction of coagulation factors, leading
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SummaryBlood platelets play a crucial role in the response of blood to vascular injury through adherence to exposed subendothelial vessel wall structures, the subsequent release of substances which trigger platelet aggregation, and release-associated changes in platelet surface properties which facilitate the interaction of coagulation factors, leading
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Frontiers in Bioscience, 2003
Diabetes is characterized by impaired fibrinolysis. This phenomenon reflects augmented concentrations of plasminogen activator inhibitor type-1 in tissues and in blood. The derangement appears to depend in part on elevated concentrations of free fatty acids, triglycerides, and insulin in association with the insulin resistance syndrome.
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Diabetes is characterized by impaired fibrinolysis. This phenomenon reflects augmented concentrations of plasminogen activator inhibitor type-1 in tissues and in blood. The derangement appears to depend in part on elevated concentrations of free fatty acids, triglycerides, and insulin in association with the insulin resistance syndrome.
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La Ricerca in Clinica e in Laboratorio, 1979
This review deals with aspects of fibrinolysis in which significant developments have taken place in the last few years. The structural changes of plasminogen during its activation are now identified precisely; the recent description of a thrombotic tendency in a kindred characterized by a defect of this protein emphasizes its important role in the ...
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This review deals with aspects of fibrinolysis in which significant developments have taken place in the last few years. The structural changes of plasminogen during its activation are now identified precisely; the recent description of a thrombotic tendency in a kindred characterized by a defect of this protein emphasizes its important role in the ...
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Scandinavian Journal of Gastroenterology, 1987
The two final phases in the haemostatic process, plasma coagulation with the formation of a fibrin clot, and fibrinolysis leading to the dissolution of fibrin clots, are reviewed. Coagulation may be initiated either by reactions occurring between components of the blood alone, the intrinsic pathway, or by reactions which also involve tissue components,
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The two final phases in the haemostatic process, plasma coagulation with the formation of a fibrin clot, and fibrinolysis leading to the dissolution of fibrin clots, are reviewed. Coagulation may be initiated either by reactions occurring between components of the blood alone, the intrinsic pathway, or by reactions which also involve tissue components,
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Scandinavian Journal of Haematology, 1969
A clinical series of 40 patients was observed for 5 months, in the course of which every patient took 1.5 g. metformin daily for 3 months. No increase in fibrinolytic activity was demonstrable by euglobulin lysis test during the course of treatment. Nor did the fibrinogen and cholesterol values change. Blood glucose decreased.
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A clinical series of 40 patients was observed for 5 months, in the course of which every patient took 1.5 g. metformin daily for 3 months. No increase in fibrinolytic activity was demonstrable by euglobulin lysis test during the course of treatment. Nor did the fibrinogen and cholesterol values change. Blood glucose decreased.
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Nature, 1953
THE plasma of normal people has been shown to contain a labile, active fibrinolysin1,2,3. Since this enzyme is not necessarily plasmin and in some ways behaves differently from plasmin, it has been tentatively termed ‘plasma active fibrinolysin’2. The enzyme disappears rapidly from fluid blood and plasma kept at room temperature; but if blood is cooled
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THE plasma of normal people has been shown to contain a labile, active fibrinolysin1,2,3. Since this enzyme is not necessarily plasmin and in some ways behaves differently from plasmin, it has been tentatively termed ‘plasma active fibrinolysin’2. The enzyme disappears rapidly from fluid blood and plasma kept at room temperature; but if blood is cooled
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