Results 71 to 80 of about 11,684,155 (300)

p190A/ARHGAP35 and p190B/ARHGAP5 proteins in endometrial cancer: a novel cancer‐relevant paralog interplay

open access: yesMolecular Oncology, EarlyView.
This study identifies ARHGAP5, in addition to the frequently mutated ARHGAP35, as significantly mutated in endometrial cancer. Mutations in both genes co‐occur and are associated with their correlated downregulation. Functional CRISPR studies show that both paralogs regulate similar pathways, including actin cytoskeleton organization.
Mathilde Pinault   +12 more
wiley   +1 more source

Editorial: Financial Technology (Fintech) and Sustainable Financing, 3rd Edition

open access: yes
It is an honor to serve as the Guest Editor for this Special Issue, “Financial Technology (Fintech) and Sustainable Financing [...
Sisira Colombage
core   +1 more source

Effects of IGFBP4 deficiency on human preadipocyte proliferation and differentiation through the IGF1R/AKT pathway

open access: yesFEBS Open Bio, EarlyView.
IGFBP4 knockdown (KD) impairs preadipocyte proliferation and is associated with IGF1R protein downregulation and attenuated AKT phosphorylation. The mechanisms by which IGFBP4 KD influences the IGF1R/AKT signaling pathway involve newly synthesized proteins and lysosomal degradation pathways. Created in BioRender.
Yujia Guo   +6 more
wiley   +1 more source

Financial risk tolerance: An analysis of unexplored factors [PDF]

open access: yes, 2013
Using data from a survey alliance between Kiplinger's Personal Finance Magazine, PBS's Nightly Business Report, and FinaMetrica, this study explores various demographical and attitudinal factors related to financial risk tolerance.
Gibson, RJ   +2 more
core  

Information and Communication Technology Adoption and Life Insurance Market Development: Evidence from Sub-Saharan Africa

open access: yes, 2022
As part of the Fourth Industrial Revolution (4IR), blockchain, fintech (financial technology), and insurtech (insurance technology) are some innovations that have been rolled out in the financial landscape and have captured the imaginations of ...
Sibindi, Athenia Bongani   +1 more
core   +1 more source

Treatment with KCL‐286, a first‐in‐class retinoic acid receptor‐β (RARβ) agonist, ameliorates neuronal DNA damage and inflammation in a mouse model of Alzheimer's disease

open access: yesFEBS Open Bio, EarlyView.
Repair of neuronal DNA damage in Alzheimer's disease by KCL‐286. (A) Amyloid‐β oligomers and plaques impair neuronal DNA repair pathways, leading to DNA double‐strand breaks and glial activation. (B) KCL‐286 activates RARβ/RXR signalling via retinoic acid response elements (RAREs), associated with increased BRCA1 expression, enhanced DNA repair and ...
Natasha Hill   +6 more
wiley   +1 more source

Swinburne University of Technology Statutory and Financial Report 2005

open access: yes, 2006
Swinburne University of Technology Statutory and Financial Report ...
Swinburne University of Technology
core   +1 more source

Buddhism in the Blockchain Age: An Ethical Analysis of Cryptocurrency

open access: yesJournal of International Buddhist Studies
Like all religious traditions, Buddhism must continually respond to emerging technologies while remaining grounded in its ethical and philosophical foundations.
Billy Wheeler
doaj   +1 more source

Pathways and pitfalls: a qualitative study of student experiences in biomedical science education

open access: yesFEBS Open Bio, EarlyView.
Biomedical science students from underrepresented backgrounds face barriers including financial strain, disrupted laboratory access and cultural exclusion. Peer networks provide vital support when institutional systems are difficult to navigate. To create inclusive learning environments and achieve academic success, educators should blend active, hands‐
Olivia J. Russell   +8 more
wiley   +1 more source

Swinburne University of Technology Statutory and Financial Report 2006

open access: yes, 2007
Swinburne University of Technology Statutory and Financial Report ...
Swinburne University of Technology
core   +1 more source

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