BMI‐1 modulation and trafficking during M phase in diffuse intrinsic pontine glioma
The schematic illustrates BMI‐1 phosphorylation during M phase, which triggers its translocation from the nucleus to the cytoplasm. In cycling cells, BMI‐1 functions within the PRC1 complex to mediate H2A K119 monoubiquitination. Following PTC596‐induced M phase arrest, phosphorylated BMI‐1 dissociates from PRC1 and is exported to the cytoplasm via its
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Zoonotic nematode larvae in Greenland halibut (<i>Reinhardtius hippoglossoides</i>) from Greenlandic waters: Occurrence, fillet distribution and association with Mushy halibut syndrome. [PDF]
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Linking brain and behavior states in Zebrafish Larvae locomotion using hidden Markov models. [PDF]
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iFish: a comprehensive multi-omics database for fish genetics, transcriptional regulation, and epigenetic research. [PDF]
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Biofortification of Sea Bream Fillets with Artichoke Polyphenols: Effects on Antioxidant Capacity, Shelf Life, and Sensory Quality. [PDF]
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Barriers Broken: Genetic swamping in restored Brook Trout populations. [PDF]
Smith RJ, Kulp MA, Fitzpatrick BM.
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