Results 101 to 110 of about 708,864 (357)

Effect of Topical Insulin on Diabetic Wound Healing among Patients with Diabetic Foot Ulcer [PDF]

open access: yesEgyptian Journal of Health Care, 2019
Diabetic foot ulcer is a debilitative disease with severe consequences in diabetic patients. Woundhealing is a complex biological process that restores skin integrity. Conventional dressings werefound ineffective for these ulcers.
Hoda Abdou Abd El-Monem El-Deeb   +1 more
doaj   +1 more source

A Microbial Lipid‐ATP Synthase Axis Fuels NK Cell Antitumor Activity

open access: yesAdvanced Science, EarlyView.
This study focuses on the mechanism by which gut microbiota‐derived outer membrane vesicles (OMVs) regulate NK cell antitumor activity. B. intestinalis is identified to decrease extra‐intestinal tumor growth via its OMVs enriched in sphingosine (SP).
Kaiyuan Yu   +16 more
wiley   +1 more source

Ulcer-free survival following management of foot ulcers in diabetes

open access: yes, 2012
AIMS: Measures of healing rate may not give a complete indication of the effectiveness of overall management of diabetic foot ulcers. Apart from healing and speed of healing, the outcomes of greatest importance to the patient are avoidance of any ...
Susan Chipchase (17155477)   +4 more
core   +1 more source

The Impact of Diabetic Foot Ulcer on Health Related Quality Of Life and Employment among Diabetics Attending Tertiary Care Teaching Hospital, Davangere

open access: yesNational Journal of Community Medicine, 2018
Background: Diabetic foot ulcer is the most common complication of diabetes mellitus. Diabetic foot disease reduces quality of life. It is associated with early retirement and reduced productivity. This research was conducted to study the health related
Lekha Tejaswi Y, Navinkumar Angadi
doaj  

Queensland’s high risk foot database: tracking the length and width of Queensland’s foot ulcers [PDF]

open access: yes, 2013
Background\ud Foot ulcers are a leading cause of avoidable hospital admissions and lower extremity amputations. However, large clinical studies describing foot ulcer presentations in the ambulatory setting are limited.
Kamp, Maarten C   +13 more
core   +1 more source

Inflammation Unchecked: Concurrent Kawasaki Disease and Stevens‐Johnson Syndrome in an 18‐Month‐Old Child

open access: yes
Arthritis Care &Research, EarlyView.
Catherine Deffendall   +6 more
wiley   +1 more source

A Keratinocyte‐Mast Cell NF‐κB2/CXCL2/IL‐6 Amplification Loop Enhances Cutaneous Antifungal Defense Against C. albicans

open access: yesAdvanced Science, EarlyView.
ABSTRACT Mast cells (MCs), key innate immune sentinels at the host–environment interface, serve as primary responders to invading pathogens. However, their specific contribution to host defense against cutaneous Candida albicans (C. albicans) infection and their synergy with other immune and non‐immune cells remain poorly understood. Here, we show that
Yan Yuan   +12 more
wiley   +1 more source

Risk factors associated with the recurrence of diabetic foot ulcers: A meta-analysis.

open access: yesPLoS ONE
AimsDiabetic foot ulcers have caused significant medical, economic and social consequences for patients, families and society. With appropriate treatment, many diabetic foot ulcers can heal, temporarily avoiding possible amputation.
Chunmei Lin   +4 more
doaj   +1 more source

Does intensive glycaemic control promote healing in diabetic foot ulcers? – a feasibility study

open access: yesBMJ Open, 2020
Introduction One in four diabetes patients will develop a foot ulcer over their lifetime. The role of glycaemic control in the healing of foot ulcers in diabetes patients is not supported by randomised controlled trial (RCT) data.Objectives To determine ...
Ajith Dissanayake   +6 more
doaj   +1 more source

m6A‐Mediated Glycolysis by IL‐37 Drives T Cell Metabolic Reprogramming to Regulate Colitis

open access: yesAdvanced Science, EarlyView.
This study identifies an IL‐37/SIGIRR‐METTL14 regulatory axis that suppresses global m6A modification in CD4+ T cells. IL‐37 signaling, mediated through SIGIRR, inhibits IRAK4 and JNK phosphorylation, leading to downregulation of the methyltransferase METTL14.
Xiaoyan Wang   +26 more
wiley   +1 more source

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