Results 161 to 170 of about 77,789 (337)
Is furosemide a nephrotoxic for hospitalized patients? [PDF]
Kyaw Kyaw Hoe +2 more
openalex +1 more source
Potentiometric determination of furosemide
A simple method is presented for the potentiometric determination of small quantities of furosemide in a pure state and in pharmaceutical formulations, using 0.1 mol l-1 chloramine-T as titrant.
Nikolić, Kosta, Medenica, Mirjana
core
We assessed the effects of hypertonic saline solution (HSS) plus furosemide versus furosemide alone in patients with acute decompensated heart failure (ADHF).
Hernandez, Adrian V. +5 more
core +1 more source
Furosemide Induced Tubulointerstitial Nephritis
Introduction Acute interstitial nephritis (AIN), also called tubulointerstitial nephritis, is a renal pathology that can cause a significant decline in kidney function. Drug-induced AIN accounts for 70% of all cases and is often due to non-steroidal anti-
Joseph, David +4 more
core
Aims Prescribing is a complex, essential skill that doctors must acquire to practice medicine safely and effectively. The British Pharmacological Society has historically provided a core curriculum to guide clinical pharmacology and prescribing education in UK medical schools.
Dagan O. Lonsdale +5 more
wiley +1 more source
HYPOTONIC SALINE AND FUROSEMIDE DIURESIS IN BARTTER'S SYNDROME [PDF]
Edward C. Kohaut +2 more
openalex +1 more source
Postoperative fluid overload is associated with increased mortality and morbidity in infants with congenital heart disease (CHD). Loop diuretics, such as furosemide, are commonly used to prevent fluid overload in the postoperative period.
Ayşegül Aşır +2 more
doaj +1 more source
Does Albumin Preinfusion Potentiate Diuretic Action of Furosemide in Patients with Nephrotic Syndrome? [PDF]
Ki Young Na +10 more
openalex +1 more source
Severe Lactic Acidosis in Decompensated Cirrhosis Despite Nondiagnostic Imaging
ABSTRACT Lactic acidosis is a strong predictor of mortality in cirrhosis, reflecting both impaired hepatic clearance and systemic tissue hypoxia. We describe a 38‐year‐old man with decompensated alcohol‐associated cirrhosis who developed severe lactic acidosis despite stable hemodynamics and initially nondiagnostic vascular imaging.
Nakul Ganju +5 more
wiley +1 more source

