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Incorporation of galactose into galactosyltransferase
Biochimica et Biophysica Acta (BBA) - General Subjects, 1977Bovine skim milk galactosyltransferase (EC 2.4.1.22) retained its catalytic activity after partial enzymatic removal of sialic acid and galactose. Desialylated and degalactosylated galactosyltransferase was a galactosyl acceptor in the galactosyltransferase reaction. [14C]Galactose from UDP-[14C]galactose was incorporated into the carbohydrate-depleted
C R, Geren +3 more
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CONCENTRATION OF GALACTOSE BY LEUCOCYTES
Canadian Journal of Biochemistry and Physiology, 1963Leucocytes suspended in native plasma took up galactose and held it within the cells at a concentration 3–6 times greater than that in the plasma. Conversion to galactose-1-phosphate, uridinediphosphogalactose, and other, unidentified compounds occurred. The accumulation of galactose was not impaired in diabetes, and was not influenced by insulin.
N, KALANT, R, SCHUCHER
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Mutations in the galactose-operator
Molecular and General Genetics MGG, 1970Constitutive mutations in the galactose operator in E. coli arise with a frequency ten times smaller than in the regulator gene. The operator constitutive mutations do not arise as a consequence of mutagen treatment. Operator constitutive mutations do not revert to wildtype spontaneously or after mutagen treatment.
L, Fiethen, P, Starlinger
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Intestinal absorption of galactose
Biochimica et Biophysica Acta, 1960Abstract Galactose absorption from the rat intestine was studied in the intact animal, in the perfused intestine and in the isolated intestinal loop. In the intact animal an average porto-arterial difference of 16.1 milligram per cent galactose was found. At the same time there was no porto-arterial difference in the total reducing sugar.
R, TZUR, B, SHAPIRO
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2000
Abstract The impact of galactose toxicity depends on the particular pathway of galactose metabolism present in various cells and the structure of the tissues and organs containing these cells. Experimental studies using high-galactose diets have demonstrated toxic complications in tissues of the eye, liver, kidney, gonads, and ...
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Abstract The impact of galactose toxicity depends on the particular pathway of galactose metabolism present in various cells and the structure of the tissues and organs containing these cells. Experimental studies using high-galactose diets have demonstrated toxic complications in tissues of the eye, liver, kidney, gonads, and ...
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Galactose 1-phosphate in galactose cataract
Biochimica et Biophysica Acta, 1955Y, SCHWARZ, L, GOLBERG
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Journal of the American Medical Association, 1958
Galactosemia and galactosuria characterize a hereditary disease caused by an inborn error of metabolism, which produces in an affected child the inability to metabolize galactose and lactose normally. Some appropriate terms that have been suggested and used synonymously are galactose diabetes, essential galactosuria, congenital galactosemia, congenital
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Galactosemia and galactosuria characterize a hereditary disease caused by an inborn error of metabolism, which produces in an affected child the inability to metabolize galactose and lactose normally. Some appropriate terms that have been suggested and used synonymously are galactose diabetes, essential galactosuria, congenital galactosemia, congenital
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Galactose-I-Phosphoric Acid in Galactose Metabolism
Nature, 1939The synthesis of α (?)-galactose-l-phosphoric acid has provided a means of examining the constitution of the galactosephosphate accumulating in the liver during galactose assimilation1. Both natural and synthetic esters are non-reducing, very readily hydrolysed by acid, and resistant to alkaline hydrolysis.
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1979
The hypothesis of catabolite repression predicts that energy sources other than glucose should repress glucose-sensitive enzymes in cells grown in media that limit their ability to utilize the catabolites rapidly for the synthesis of macromolecules; and in fact, partial amino acid, purine, pyrimidine, or phosphate starvation has this effect.
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The hypothesis of catabolite repression predicts that energy sources other than glucose should repress glucose-sensitive enzymes in cells grown in media that limit their ability to utilize the catabolites rapidly for the synthesis of macromolecules; and in fact, partial amino acid, purine, pyrimidine, or phosphate starvation has this effect.
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