Results 151 to 160 of about 27,415 (188)
Evolution of intrinsically disordered regions in vertebrate galectins for phase separation. [PDF]
Lin YH, Chen YC, Sun YC, Huang JR.
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Multiplexed targeted mRNA profiling of alcohol-related liver disease reveals stage-specific dysregulation of signaling pathways. [PDF]
Rasic D +4 more
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Abnormal Galectin Signaling in the Pathomechanisms of Placental Dysfunction in Gestational Diabetes Mellitus. [PDF]
Szukiewicz D.
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A novel circulating osteoimmunological signature for diagnosis: integrating CXCL2, FYN, galectin-3, and STING in postmenopausal osteoporosis. [PDF]
Ma S, Yao Q, Bao X, Li Y.
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101P Galectin 3 binding protein signature as early detection biomarker of glioma
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Revealing the Identity of Human Galectin-3 as a Glycosaminoglycan-Binding Protein
2022Human galectin-3 (Gal-3) is a β-galactoside-binding lectin. This multitasking protein preferentially interacts with N-acetyllactosamine moieties on glycoconjugates. Specific hydroxyl groups (4-OH, 6-OH of galactose, and 3-OH of glucose/N-acetylglucosamine) of lactose/LacNAc are essential for their binding to Gal-3.
Jared L, Edwards +3 more
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Galectin-3 Is a Nuclear Matrix Protein Which Binds RNA
Biochemical and Biophysical Research Communications, 1995The endogenous galectin-3 is a carbohydrate-binding protein of M(r) approximately 30,000 serving in the cytoplasm and on the cell surface as a receptor for ligands containing poly-N-acetyllactosamine sequences. In addition, galectin-3 has been demonstrated to be associated in the nucleus with ribonucleoprotein complexes and to act as a pre-mRNA ...
L, Wang, H, Inohara, K J, Pienta, A, Raz
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Clinical and Experimental Rheumatology, 2021
Venous (VTE) and arterial (AT) thrombosis in systemic lupus erythematosus (SLE) are poorly explained and difficult to predict. Leptin and tumour necrosis factor-like weak inducer of apoptosis (TWEAK) have been linked to subclinical atherosclerosis and galectin-3-binding protein (G3BP) to type I interferon activation and a pro-thrombotic environment ...
Peretz, Anne Sofie R. +4 more
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Venous (VTE) and arterial (AT) thrombosis in systemic lupus erythematosus (SLE) are poorly explained and difficult to predict. Leptin and tumour necrosis factor-like weak inducer of apoptosis (TWEAK) have been linked to subclinical atherosclerosis and galectin-3-binding protein (G3BP) to type I interferon activation and a pro-thrombotic environment ...
Peretz, Anne Sofie R. +4 more
openaire +3 more sources

