Results 21 to 30 of about 106,721 (258)

Anti-GBM glomerulonephritis involves IL-1 but is independent of NLRP3/ASC inflammasome-mediated activation of caspase-1 [PDF]

open access: yes, 2011
IL-1b and IL-18 are proinflammatory cytokines that contribute to renal immune complex disease, but whether IL-1b and IL-18 are mediators of intrinsic glomerular inflammation is unknown.
Volker Vielhauer   +47 more
core   +2 more sources

Combination Therapy With Rituximab and Cyclophosphamide for Remission Induction in ANCA Vasculitis

open access: yesKidney International Reports, 2018
Remission induction in antineutrophil cytoplasmic autoantibody (ANCA) vasculitis may be complicated by slow response to treatment and toxicity from glucocorticoids.
Frank B. Cortazar   +6 more
doaj   +1 more source

The impact of reclassification of C3 predominant glomerulopathies on diagnostic accuracy, outcome and prognosis in patients with C3 glomerulonephritis

open access: yesBMC Nephrology, 2020
Background C3 glomerulonephritis is a recently described entity with heterogeneous histopathological features. This study was conducted to assess the effect of reclassification of C3 glomerulopathies on renal outcomes, mortality, and response to therapy.
P. Puri   +5 more
doaj   +1 more source

Effect of dsRNA on Mesangial Cell Synthesis of Plasminogen Activator Inhibitor Type 1 and Tissue Plasminogen Activator [PDF]

open access: yes, 2009
Background/Aims: Viral infections are a major problem worldwide and many of them are complicated by virally induced glomerulonephritides. Progression of kidney disease to renal failure is mainly attributed to the development of renal fibrosis ...
Wörnle, Markus   +4 more
core   +1 more source

Recurrent Crescentic Infection Related Glomerulonephritis: a rare presentation

open access: yesIndian Journal of Kidney Diseases, 2022
Infection Related Glomerulonephritis (IRGN) is an immune-mediated disease caused by non-renal infections. The most common histopathology is diffuse endocapillary proliferative glomerulonephritis with neutrophilic infiltrate. IRGN presenting as crescentic
Kapil Naveen Sejpal   +4 more
doaj   +1 more source

Etiopathogenetic, Morphological, Diagnostic and Therapeutic Aspects of Acute Glomerulonephritis: Current Status

open access: yesАрхивъ внутренней медицины, 2020
The review provides up-to-date information on the etiological factors and pathogenic mechanisms of development, morphological changes, clinical and laboratory manifestations of acute glomerulonephritis, as well as therapeutic possibilities for its ...
I. T. Murkamilov   +4 more
doaj   +1 more source

Clinicopathological Features and Kidney Survival in Rapidly Progressive Glomerulonephritis: A Single-Center Experience

open access: yes, 2022
© 2022 Turkish Society of Nephrology. All rights reserved.Objective: Advanced age, need for dialysis and high creatinine values at the time of admission, anti-glomerular basement membrane antibody positivity, pulmonary hemorrhage, and fibrous crescents ...
Aylı, Mehmet Deniz   +17 more
core   +1 more source

Influence of infiltrate density in the interstitium on the prognosis of primary glomerulonephritis [PDF]

open access: yesVojnosanitetski Pregled, 2019
Background/Aim. Development of inflammatory changes, fibrosis and loss of morphological structures of the interstitium have an important role in pathogenesis of primary glomerulonephritis, affecting the development, course and prognosis of the disease ...
Božić Dušan   +5 more
doaj   +1 more source

Bartonella-Associated Endocarditis with Severe Active Crescentic Glomerulonephritis and Acute Renal Failure

open access: yesCase Reports in Nephrology, 2021
We report a case of severe acute kidney failure due to crescentic glomerulonephritis who presented initially with culture-negative endocarditis with vegetations on the aortic valve.
Ramanath Dukkipati   +3 more
doaj   +1 more source

Clinical periodontal diagnosis

open access: yesPeriodontology 2000, EarlyView., 2023
Abstract Periodontal diseases include pathological conditions elicited by the presence of bacterial biofilms leading to a host response. In the diagnostic process, clinical signs such as bleeding on probing, development of periodontal pockets and gingival recessions, furcation involvement and presence of radiographic bone loss should be assessed prior ...
Giovanni E. Salvi   +5 more
wiley   +1 more source

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