Results 301 to 310 of about 175,446 (337)
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The mesangium and glomerulonephritis
Klinische Wochenschrift, 1982The mesangium of the glomerular capillary ultrafilter is a specialized pericapillary tissue. In adult mammals its location is limited to the axial portions of the loop, but it extends peripherally to encircle the capillary in the fetal state and in certain glomerular diseases.
David H. Lovett +3 more
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Membranoproliferative glomerulonephritis--a new look at an old entity.
New England Journal of Medicine, 2012This review discusses the causes, pathogenesis, and clinical management of membranoproliferative glomerulonephritis, which accounts for 7 to 10% of biopsy-confirmed glomerulonephritis cases.
S. Sethi, F. Fervenza
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KDOQI US commentary on the 2012 KDIGO clinical practice guideline for glomerulonephritis.
American Journal of Kidney Diseases, 2013Glomerulonephritis (GN) is an important cause of morbidity and mortality in patients of all ages throughout the world. Because these disorders are relatively rare, it is difficult to perform randomized clinical trials to define optimal treatment for many
L. Beck +8 more
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Apoptosis and Glomerulonephritis
2005Glomerulonephritis (GN) is a form of autoimmunity in which apoptosis may be a double-edged sword. Resolution of GN can be promoted by apoptosis of infiltrating leucocytes and excess resident glomerular cells, leading to efficient anti-inflammatory clearance by macrophages and mesangial cells.
Jean-François Cailhier +3 more
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2018
Membranous glomerulonephritis (MGN) is the most common cause of adult-onset nephrotic syndrome, and a common glomerular cause of end-stage renal failure. It is caused by antibodies to podocyte surface molecules, usually autoantibodies. In most patients with primary membranous nephropathy the target is the phospholipase A2 receptor.
Daniel C. Cattran, Heather N. Reich
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Membranous glomerulonephritis (MGN) is the most common cause of adult-onset nephrotic syndrome, and a common glomerular cause of end-stage renal failure. It is caused by antibodies to podocyte surface molecules, usually autoantibodies. In most patients with primary membranous nephropathy the target is the phospholipase A2 receptor.
Daniel C. Cattran, Heather N. Reich
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On Treating Glomerulonephritis
Archives of Internal Medicine, 1979A patient seeks treatment for hematuria and proteinuria and undergoes diagnostic examination that excludes urologic lesions and does not reveal characteristic findings of specific renal diseases, such as acute poststreptococcal glomerulonephritis. To clarify further the nature of the disease, a renal biopsy is performed.
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Immunopathogenesis of Glomerulonephritis
1971Publisher Summary This chapter discusses the experimental studies related to immunopathogenesis of glomerulonephritis. The study also establishes that new nephritogenic antigens in human glomerulonephritis include those of streptococcal membranes that can be readily identified in renal biopsies from patients with acute post-streptococcal ...
F.J. DIXON, G.A. ANDRES
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1997
The various forms of human glomerulonephritis are a group of diseases of largely unknown etiology, which account for a significant proportion of human renal disease. Most have an immune pathogenesis and injury results from either immune complex deposition or delayed hypersensitivity-type reactions within the glomerulus [1].
Victoria Cattell, H. Terence Cook
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The various forms of human glomerulonephritis are a group of diseases of largely unknown etiology, which account for a significant proportion of human renal disease. Most have an immune pathogenesis and injury results from either immune complex deposition or delayed hypersensitivity-type reactions within the glomerulus [1].
Victoria Cattell, H. Terence Cook
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Epidemiology, pathogenesis, treatment and outcomes of infection-associated glomerulonephritis
Nature Reviews Nephrology, 2019A. Satoskar, S. Parikh, T. Nadasdy
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