Results 11 to 20 of about 174,209 (261)
Kufa Journal for Nursing Sciences, 2022 Background: Glucose-6-phosphate dehydrogenase deficiency is a genetic disorder that affects red blood cells, which carry oxygen from the lungs to tissues throughout the body.
Sadiq Sabhan Mosa
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The inorganic pyrophosphatase activity of erythrocytes in favism
The Turkish Journal of Pediatrics, 1961 Inorganic pyrophosphatase activity of erythrocytes was found to be decreased in subjects affected by the glucose-6-phosphate dehydrogenase deficiency. During the acute hemolytic episode of favism, pyrophosphatase activity shows a further critical fall.
Güngör Ernişli, Faolo Brunetti
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Advances in Human Biology, 2021 Introduction: Hyperbilirubinemia is a common complication among neonates. The objective of this cross-sectional study was the prevalence of glucose 6-phosphate dehydrogenase (G6PD) deficiency among neonates with hyperbilirubinemia in the west of Iran ...
Fatemeh Eghbalian +3 more
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Glucose-6-phosphate dehydrogenase deficiency in Nigerian children. [PDF]
PLoS ONE, 2013 Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most common human enzymopathy and in Sub-Saharan Africa, is a significant cause of infection- and drug-induced hemolysis and neonatal jaundice.
Olatundun Williams +5 more
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Fulminant hemolysis in glucose‐6‐phosphate dehydrogenase deficiency
Clinical Case Reports, 2018 Key Clinical Message Glucose‐6‐phosphate dehydrogenase (G6PD) deficiency is an X‐linked disorder affecting some 400 million people worldwide. Though clinically silent, it may result in hemolysis on oxidative stress induced by drugs or infections.
Bushra Moiz, Sidra Asad Ali
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MOLECULAR BASIS OF G6PD DEFICIENCY: CURRENT STATUS AND ITS PERSPECTIVE [PDF]
Acta Medica Iranica, 2008 Glucose-6-phosphate dehydrogenase is an essential enzyme to cell growth. Its deficiency of enzyme plays an important role in senescence and death signaling.
M. R. Noori-Daloii, M. Daneshpajooh
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Journal of Lipid Research, 1973 Cyclopropenoid fatty acids in the diet of rainbow trout caused significant reductions in liver protein and activity of glucose-6-phosphate dehydrogenase, NADP-linked isocitrate dehydrogenase, lactate dehydrogenase, and malate dehydrogenase.
S.L. Taylor, M.W. Montgomery, D.J. Lee
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Experimental Biology and Medicine, 1968 SummaryThe utilization of glucose and other nonphosphorylated sugars by partially purified normal G-6-PD and G-6-PD Mediterranean was investigated. Both enzymes have the capacity to oxidize glucose with a Km of approximately 0.8 M. The utilization of glucose by both enzymes was markedly stimulated by the presence of bicarbonate, and the activity with ...
C, Kissin, E, Beutler
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Journal of Medical Case Reports, 2008 Introduction Glucose-6-phosphate dehydrogenase deficiency is an X-linked recessive disease that causes acute or chronic hemolytic anemia and potentially leads to severe jaundice in response to oxidative agents.
Cossio de Gurrola Gladys +7 more
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Glucose-6-Phosphate Dehydrogenase Variants in Greeks
Human Heredity, 1970 On a sample of the Greek population, composed of 708 adult individuals (420 men and 288 women), G6PD types were studied by means of vertical electrophoresis. In addition to the common B type, three variants of the enzyme were found: (a) In one female, the rapidly migrating A type, the first to be described in a Caucasian subject; (b) In two males and ...
Angelopoulos, B.T., Delitheos, A.K.
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