Results 31 to 40 of about 174,209 (261)
Glucose-6-phosphate Dehydrogenase Deficiency
Clinical Pediatric Hematology-Oncology, 2007 Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most common enzyme disorder. There are more than 400 million people worldwide with G6PD deficiency, and its distribution is similar to that of malaria. G6PD deficiency is an X-linked recessive disorder. Most patients with G6PD deficiency may be asymptomatic throughout their lives.
openaire +1 more source
A yeast model of 5‐oxoproline accumulation reveals a general toleration to 5‐oxoproline
FEBS Open Bio, EarlyView.Using a yeast model, we show that even high accumulation of 5‐oxoproline causes only mild cellular stress and does not trigger oxidative stress. Instead, cells adapt by activating efflux pumps and diverse protective pathways, suggesting that previously proposed harmful effects of 5‐oxoproline may arise from indirect metabolic imbalances rather than the
Pratiksha Dubey +4 more
wiley +1 more source
Quantitative study of erythrocyte glucose-6-phosphate dehydrogenase
Siriraj Medical Journal, 2023 The method and result of erythrocyte glucose-6-phosphate dehydrogenase (G6PD) assay in Thai subjects are presented. In 266 males the distribution of G6PD values is obviously bimodal but not absolutely separated into two groups.
Vicharn Panich, Prawase Wasi
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FEBS Open Bio, EarlyView.Abruptly changing from aerobic to anaerobic conditions (sudden anaerobization) induced growth inhibition and a significant increase in intracellular labile ferrous iron in the aerotolerant anaerobe Amphibacillus xylanus. We found that free flavins mediate efficient electron transfer from NADH to ferric iron under anaerobic conditions, suggesting that ...
Shinya Kimata +13 more
wiley +1 more source
Glucose-6-Phosphate Dehydrogenase (G6PD) deficiency
Majallah-i Dānishgāh-i ̒Ulūm-i Pizishkī-i Bābul, 2006 .
Y ZahedPasha, M Ahmad Pour, A ZahedPasha
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FEBS Open Bio, EarlyView.Time‐restricted feeding (TRF) in mice increased liver fatty acid oxidation and decreased fatty acid biosynthesis. These alterations persisted when TRF was discontinued and the host was infected with Mycobacterium tuberculosis. Pre‐exposure to TRF did not alter tissue (lung and spleen) mycobacterial burden but significantly reduced CD3+ T cells in lungs
Ashish Gupta +7 more
wiley +1 more source
Pakistan Armed Forces Medical Journal, 2010 Objective: To determine the frequency of G6PD deficiency in asymptomatic Pakistan population. Study Design: Cross sectional study. Place and Duration of study: This was carried out at Pakistan Air Force (PAF) Hospital at PAF Base Masroor Karachi between
Mehmood Aamer +2 more
doaj +2 more sources
Glucose 6 phosphate dehydrogenase as a cause of neonatal jaundice
The Turkish Journal of Pediatrics, 1963 A case of neonatal hyperbilirubinemia is reported. Tests indicated a glucose 6 phosphate dehydrogenase deficiency as the cause of the hemolytic disease. The authors have undertaken an intensive study of the incidence of G-6-PD deficiency in Turkey.
Burhan Say +3 more
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GelMA‐based 3D spheroids recapitulate transcriptomic and functional hallmarks of myeloid sarcoma
FEBS Open Bio, EarlyView.GelMA 5% hydrogels support the formation of myeloid leukemia spheroids that recapitulate MS‐specific features, including G1 arrest, apoptosis, and ECM‐driven transcriptomic reprogramming. The 3D model mimicked soft‐tissue‐like stiffness and oxygen conditions, and transcriptomic convergence with primary MS samples confirmed its utility as a preclinical ...
Nicolas Germain +11 more
wiley +1 more source
Glucose-6-Phosphate Dehydrogenase Deficiency and Neonatal Hyperbilirubinemia [PDF]
Iranian Journal of Neonatology, 2015 Background: Jaundice is affecting over 60-80 percent of neonates in the first week of life. Glucose-6-phosphate dehydrogenase (G6PD) deficiency, which is an important cause of pathologic hyperbilirubinemia, can lead to hemolytic anemia, jaundice and ...
Ezzat Khodashenas +4 more
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