Results 291 to 300 of about 171,724 (348)
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Diabetes Care, 1981
Glucose tolerance is known to decrease with advancing age. This decline begins in the third or fourth decade of life and is progressive throughout the entire adult life span. The primary cause of this age-related impairment in glucose metabolism results from tissue unresponsiveness to insulin.
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Glucose tolerance is known to decrease with advancing age. This decline begins in the third or fourth decade of life and is progressive throughout the entire adult life span. The primary cause of this age-related impairment in glucose metabolism results from tissue unresponsiveness to insulin.
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Diabetic glomerulosclerosis without glucose intolerance
The American Journal of Medicine, 1975The pathophysiology of the microangiopathy of diabetes mellitus is poorly understood, and the relevance of carbohydrate intolerance remains uncertain. Four patients are presented with renal abnormalities suggestive of diffuse diabetic glomeruloscierosis. These patients have no evidence of carbohydrate intolerance by standard clinical technics.
D A, Nash +3 more
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Hypophosphatemia and Glucose Intolerance
1982Hypophosphatemia is frequently observed in a variety of disease states including ketoacidosis, chronic alcoholism, malabsorption, severe burns, hyperparathyroidism, renal tubular defects, and re-feeding after starvation (1, 2). Since phosphate is a ubiquitous anion involved in such diverse functions as maintaining the structural integrity of the cell ...
D, Simonson, R A, DeFronzo
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Glucose Intolerance in Cystic Fibrosis
New England Journal of Medicine, 1969Abstract Moderate to severe oral glucose intolerance was observed in 42 per cent of 31 patients with cystic fibrosis of the pancreas. Serum insulin concentrations after glucose were low in all patients regardless of glucose tolerance. When glucagon or tolbutamide was infused at the height of the reduced insulin response to oral glucose, serum insulin ...
S, Handwerger +5 more
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Glucose intolerance in parkinson's disease
Journal of Chronic Diseases, 1974Abstract Oral glucose tolerance was found to be impaired in a large percentage of an unselected group of patients with Parkinson's Disease. The elevation of blood sugar was unrelated to patient age, duration of Parkinsonism, or degree of disability. Blood sugar levels were higher than would be expected for a random population of similar age, and 52.4
I J, Lipman, M E, Boykin, R E, Flora
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Glucose intolerance in liver cirrhosis
Metabolism, 1982Abstract Glucose intolerance and hyperinsulinemia frequently occur in patients with chronic liver failure. To investigate the importance of glucose counterregulating factors, an oral glucose tolerance test was performed on 18 patients with compensated liver cirrhosis, matched for liver function tests and degree of portal hypertension, and 10 healthy ...
RIGGIO, Oliviero +12 more
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Glucose intolerance in sarcoma patients
Cancer, 1984Twenty-seven otherwise healthy patients with localized sarcoma were examined to determine if glucose intolerance can be detected before the appearance of clinical signs of cachexia. No patient had lost weight or demonstrated severe malnutrition. Fasting plasma samples for glucose, insulin, glucagon, and free fatty acids (FFA) were obtained, and a ...
J A, Norton +4 more
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Glucose Intolerance with Atypical Antipsychotics
Drug Safety, 2002Previous studies have suggested that the atypical antipsychotics clozapine and olanzapine may be associated with an increased risk of glucose intolerance and diabetes mellitus. Early studies have also suggested an association between use of conventional antipsychotics and the development of glucose intolerance.To examine quantitatively the association ...
Karin, Hedenmalm +4 more
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Glucose Intolerance in Hypernatremic Rats
Diabetes, 1968A study has been made of the effect of hypernatremia on carbohydrate metabolism in the rat. It was shown that rats rendered hypernatremic by subcutaneous injections of hypertonie sodium solutions manifest increased fasting blood glucose levels, as well as a decreased ability to dispose of an exogenous glucose load. The mechanism by which
M, Nitzan, S, Zelmanovsky, P, Tikva
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