Results 221 to 230 of about 36,080 (282)
Correction: Novel small molecule derivatives improve survivability in the cellular model of Huntington's disease <i>via</i> improving mitochondrial fusion. [PDF]
Kodam P +5 more
europepmc +1 more source
Changes in Italian Nurses' Genetic Knowledge and Perceptions Over a Decade
ABSTRACT Genetic knowledge is increasingly important in nursing, yet often seen as of limited relevance. This study examines Italian nurses' genetic knowledge and perceived relevance, comparing current findings with data from a 2011 survey. A cross‐sectional survey was carried out using a self‐administered questionnaire. A total of 504 nurses (95.1% of
Lea Godino +3 more
wiley +1 more source
Preserved Thermoregulation in Huntington's Disease: Insights from an Observational Case-Control Study. [PDF]
Simón-Vicente L +7 more
europepmc +1 more source
Huntington's Disease Revealed by Familial Cervical Dystonia
Daniela Andriuta +3 more
openalex +2 more sources
Rats under mild caloric restriction (85% vs. ad libitum) for 2 weeks show metabolic and structural adaptations in iBAT. Caloric restriction increases glycolysis and triglyceride deposition in the tissue, leading to larger lipid droplets. It also reduces mitochondrial iBAT content, while maintaining iBAT thermogenic potential thanks to an increased ...
Giuliana Panico +11 more
wiley +1 more source
Dysregulation of huntingtin interacting protein networks in human juvenile Huntington's disease brain. [PDF]
Podvin S +5 more
europepmc +1 more source
Parabiosis, Assembloids, Organoids (PAO)
This review evaluates parabiosis, organoids, and assembloids as complementary disease models spanning systemic, organ, and multi‐organ levels. It highlights their construction strategies, applications, and current limitations, while emphasizing their integration with frontier technologies such as artificial intelligence, organ‐on‐a‐chip, CRISPR, and ...
Yang Hong +5 more
wiley +1 more source
Profiling Swallowing Safety and Physiology in People With Huntington's Disease. [PDF]
Raines C, Clark M, Donohue C.
europepmc +1 more source
MS436 competitively binds to the BD1 domain of Brd4, thereby suppressing Brd4 induced degradation of tight junction proteins via the Rnf43‐Fzd4‐β‐catenin signaling pathway. Consequently, this attenuation of degradation reduces blood‐brain barrier leakage, leading to an improved overall prognosis after stroke.
Chenxiao Li +10 more
wiley +1 more source

