Results 211 to 220 of about 114,297 (258)
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Bile Acid Clearance in Sheep with Hereditary Hyperbilirubinemia
American Journal of Veterinary Research, 1979SUMMARY The disappearance of iv injected [24-14C]cholic acid from plasma was studied in normal and mutant Corriedale and Southdown sheep exhibiting hereditary defects in hepatic organic anion transport. Hepatic cholic acid clearance was determined from the integral of the 40-minute disappearance curves fit to the sums of two exponential functions ...
L R, Engelking, R, Gronwall
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Gilbert Syndrome Increasing Unconjugated Hyperbilirubinemia in a Child With Hereditary Spherocytosis
Journal of Pediatric Hematology/Oncology, 2012Hemolytic anemia usually gives rise to only a modest elevation of serum bilirubin. Unconjugated hyperbilirubinemia of an extreme degree should raise suspicion of additional factors. We describe a 10-year-old child suffering from hereditary spherocytosis, who had unusually high levels of unconjugated serum bilirubin and was diagnosed to have Gilbert ...
Dipti, Kumar +2 more
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Molecular diagnostic update in hereditary hemolytic anemia and neonatal hyperbilirubinemia
International Journal of Laboratory Hematology, 2019AbstractHereditary hemolytic anemia (HHA) is a group of genetically and phenotypically heterogeneous disorders characterized by premature destruction of red blood cells (RBCs) with clinical manifestations ranging from asymptomatic to marked hemolytic anemia.
Anton Rets +3 more
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Bilirubin and the Genome: The Hereditary Basis of Unconjugated Neonatal Hyperbilirubinemia
Current Pharmacogenomics, 2005Severe neonatal unconjugated hyperbilirubinemia, with the risk of bilirubin encephalopathy or kernicterus in severe, untreated cases, occurs when bilirubin production exceeds the body's ability to eliminate it. The causes of neonatal hyperbilirubinemia are multifactorial and comprise increased hemolysis on the one hand, and diminished bilirubin ...
Michael Kaplan, Cathy Hammerman
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The American Journal of the Medical Sciences, 1978
Hyperbilirubinemia may be of several etiologies in the individual patient. An 18-year-old man presented with extreme hyperbilirubinemia (direct bilirubin 23.0 mg/dl, total bilirubin 60.0 mg/dl), hepatosplenomegaly, and anemia. Hematologic studies prelaparotomy documented the presence of hereditary spherocytosis.
M E, Katz, I M, Weinstein
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Hyperbilirubinemia may be of several etiologies in the individual patient. An 18-year-old man presented with extreme hyperbilirubinemia (direct bilirubin 23.0 mg/dl, total bilirubin 60.0 mg/dl), hepatosplenomegaly, and anemia. Hematologic studies prelaparotomy documented the presence of hereditary spherocytosis.
M E, Katz, I M, Weinstein
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Ukraïnsʹkij žurnal medicini, bìologìï ta sportu, 2020
Under high-intensity loads, the athlete's bodies take place a number of biochemical reactions and physiological processes that can lead to hyperbilirubinemia. The factors that can initiate the onset of this phenomenon include the syndrome of micro-damage muscle, violation of the integrity of erythrocyte membranes, decreased blood pH, malnutrition and ...
L. M. Gunina +2 more
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Under high-intensity loads, the athlete's bodies take place a number of biochemical reactions and physiological processes that can lead to hyperbilirubinemia. The factors that can initiate the onset of this phenomenon include the syndrome of micro-damage muscle, violation of the integrity of erythrocyte membranes, decreased blood pH, malnutrition and ...
L. M. Gunina +2 more
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Journal of Neurochemistry, 1980
Abstract: Immunohistochemical reactions were conducted, using the antibodies against GFA and S‐100 proteins on sections of cerebellum from the homozygous (jj) and the heterozygous (Jj) Gunn rats. Hypertrophy of the fibrous astrocytes was observed but hyperplasia of the glial cells was not. Although the molecular layer was very thin, the Bergmann fibre
K, Mikoshiba +3 more
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Abstract: Immunohistochemical reactions were conducted, using the antibodies against GFA and S‐100 proteins on sections of cerebellum from the homozygous (jj) and the heterozygous (Jj) Gunn rats. Hypertrophy of the fibrous astrocytes was observed but hyperplasia of the glial cells was not. Although the molecular layer was very thin, the Bergmann fibre
K, Mikoshiba +3 more
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Hepatology, 1985
A mutant rat strain is described with autosomal recessive conjugated hyperbilirubinemia. Transport of conjugated bilirubin and tetrabromosulfophthalein from liver to bile is severely impaired whereas uptake of organic anions from plasma to liver is normal.
Jansen, P. L. +2 more
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A mutant rat strain is described with autosomal recessive conjugated hyperbilirubinemia. Transport of conjugated bilirubin and tetrabromosulfophthalein from liver to bile is severely impaired whereas uptake of organic anions from plasma to liver is normal.
Jansen, P. L. +2 more
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Hereditary Hyperbilirubinemias: A Molecular and Mechanistic Approach
Seminars in Liver Disease, 1988P L, Jansen, R P, Oude Elferink
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[Hereditary pigmentary hepatoses (functional hyperbilirubinemias)].
Klinicheskaia meditsina, 2009Reviewed in this paper are bilirubin metabolism, definition and classification of hereditary pigmentary hepatoses, their pathogenesis and forms of inheritance, clinical picture, laboratory, instrumental and differential diagnosis, morphological changes in the hepatic tissue, and therapeutic strategies.
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