Results 161 to 170 of about 28,246 (219)
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Hypercoagulability in Athletes
Current Sports Medicine Reports, 2004Risk factors for thromboembolism are well known, and athletes are placed under conditions that can result in exposure to several of these risk factors, which include travel, trauma, immobilization, hemoconcentration, and polycythemia. Presence of a genetic hypercoagulable disorder adds additional risk.
Christopher, Meyering, Thomas, Howard
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Thalassemia and hypercoagulability
Blood Reviews, 2008Thalassemia is a congenital hemolytic disease caused by defective globin synthesis resulting in decreased quantity of globin chains. Although the life expectancy of beta-thalassemia patients has markedly improved over the last few years, patients still suffer from many complications of this congenital disease.
A. T. Taher +3 more
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HYPERTRIGLYCERIDAEMIA AND HYPERCOAGULABILITY
The Lancet, 198318 patients with severe hypertriglyceridaemia (mean fasting plasma triglyceride 5.7 mmol/l) had significantly higher concentrations of plasma fibrinogen and clotting factor Xc than did a normolipidaemic comparison group. Fibrinolytic activity was significantly lower in the hyperlipidaemic patients.
H C, Simpson +5 more
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Hypercoagulability and Thrombosis
Hematology/Oncology Clinics of North America, 1992This article has summarized known congenital and acquired alterations of hemostasis leading to thrombosis. Decreases in coagulation inhibitors, including antithrombin III, heparin cofactor II, and protein C and protein S, are of major importance in assessing patients with hypercoagulable states or patients with unexplained thrombosis.
R L, Bick, K, Ucar
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Hypercoagulability and the hypercoagulability syndromes.
American Journal of Roentgenology, 1995Hypercoagulability is a state in which an alteration of the blood shifts the hemostatic balance toward excessive platelet/fibrin deposition leading to arterial and/or venous thrombosis [1]. Although the concept of hypercoagulability has been recognized clinically for more than a century, in recent years a number of specific disorders have been define ...
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Hypercoagulability and Migraine
Headache: The Journal of Head and Face Pain, 2017Background A growing body of literature suggests that migraineurs, particularly those with aura, have an increased risk for ischemic stroke, but not via enhanced atherosclerosis. The theory that micro‐emboli induced ischemia provokes cortical spreading depression (ie, symptomatic aura) in migraineurs but ...
Gretchen E, Tietjen, Stuart A, Collins
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Hypercoagulability And Thrombosis
Medical Clinics of North America, 1994This article has stressed the common hereditary and acquired blood protein defects associated with thrombosis. The commonest hereditary defects appear to be antithrombin, protein C, and protein S deficiency, and the commonest acquired defects are anticardiolipin antibodies and the lupus anticoagulant.
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Clinics in Chest Medicine, 2010
Hypercoagulable states can be inherited or acquired. Inherited hypercoagulable states can be caused by a loss of function of natural anticoagulant pathways or a gain of function in procoagulant pathways. Acquired hypercoagulable risk factors include a prior history of thrombosis, obesity, pregnancy, cancer and its treatment, antiphospholipid antibody ...
Julia A M, Anderson, Jeffrey I, Weitz
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Hypercoagulable states can be inherited or acquired. Inherited hypercoagulable states can be caused by a loss of function of natural anticoagulant pathways or a gain of function in procoagulant pathways. Acquired hypercoagulable risk factors include a prior history of thrombosis, obesity, pregnancy, cancer and its treatment, antiphospholipid antibody ...
Julia A M, Anderson, Jeffrey I, Weitz
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Annals of Internal Medicine, 1993
To describe the major pathophysiologic mechanisms underlying inherited and secondary hypercoagulable states and to evaluate the frequency, natural history, diagnosis, and management of the various clinical disorders.Relevant clinical literature obtained from bibliographies in hematology textbooks and from computerized indexes was reviewed. A hypothesis
R L, Nachman, R, Silverstein
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To describe the major pathophysiologic mechanisms underlying inherited and secondary hypercoagulable states and to evaluate the frequency, natural history, diagnosis, and management of the various clinical disorders.Relevant clinical literature obtained from bibliographies in hematology textbooks and from computerized indexes was reviewed. A hypothesis
R L, Nachman, R, Silverstein
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1978
Many investigators have attempted to determine whether a relationship exists between excessive activity of the hemostatic mechanism and the atherosclerotic process. A major difficulty encountered in unraveling this puzzle has been the lack of reliable techniques for quantifying pertinent changes in blood coagulability.
R D, Rosenberg +3 more
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Many investigators have attempted to determine whether a relationship exists between excessive activity of the hemostatic mechanism and the atherosclerotic process. A major difficulty encountered in unraveling this puzzle has been the lack of reliable techniques for quantifying pertinent changes in blood coagulability.
R D, Rosenberg +3 more
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