Results 181 to 190 of about 4,003 (221)

Omeprazole-induced hypergastrinemia: Role of gastric acidity

Journal of Surgical Research, 1986
The increase in gastrin caused by the gastric proton pump inhibitor, omeprazole, is presumably related to inhibition of gastric acid secretion (GAS). We investigated omeprazole's effect on gastrin release by studying two doses of omeprazole which produced marked acid suppression.
G M, Larson, H W, Sullivan, P, Rayford
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Hyperacidity and hypergastrinemia following extensive intestinal resection

World Journal of Surgery, 1979
AbstractIncreased gastric acid secretion occurs after extensive intestinal resection in man, dog, rat, and monkey. Hypergastrinemia has been observed in patients with short gut syndrome and appears to accompany the hyperacidity after intestinal resection in dog, rat, and monkey. Postresectional hypergastrinemia is caused by increased release of gastrin
W C, Meyers, R S, Jones
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Does hyperparathyroidism cause hypergastrinemia?

Surgery, 1976
To define the relationship between hyperparathyroidism (HPT) and gastric function, 31 patients with HPT were prospectively studied before and 2 to 25 months after parathyroidectomy. The gastrin response to a standard test meal (STM), the basal acid output (BAO), and the peak acid output (PAO) were determined.
S D, Wilson, R B, Singh, R K, Kalkhoff
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Hypergastrinemia and the Zollinger-Ellison Syndrome

Archives of Surgery, 1972
To the Editor. —Because of the wellrecognized pitfalls inherent in overdependence upon various demonstrations of gastric hypersecretion, adaptation of radioimmunoassay techniques to the quantization of serum gastrin levels has been readily accepted as another means of diagnostic screening in patients with historical features and secretory studies ...
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Hypergastrinemia and Achlorhydria in Chronic Renal Failure

Nephron, 1985
In 68 patients with chronic renal failure (CRF), 15 patients with duodenal ulcer and 15 normal subjects, basal plasma gastrin levels and basal and stimulated gastric acid secretion were measured. Two antisera were used: antiserum R2702 with specificity for human G34 and its N-terminal fragments [G34] and antiserum 2604 with specificity for the four ...
S, Muto, N, Murayama, Y, Asano, S, Hosoda, M, Miyata   +4 more
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Hypergastrinemia of antral origin in duodenal ulcer

The American Journal of Digestive Diseases, 1978
A case of recurrent duodenal ulcer, basal gastric hypersection, and hypergastrinemia of antral origin is presented. The diagnosis was suggested preoperatively by stimulation tests with secretin and food. Billroth II antrectomy led to normalization of serum gastrin within half an hour.
C B, Lamers, C M, Ruland, H J, Joosten, H C, Verkooyen, J H, van Tongeren, J F, Rehfeld   +5 more
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Hypergastrinemia in chronic renal failure

Journal of Japanese Society for Dialysis Therapy, 1983
消化管出血は, 慢性腎不全でよく知られた合併症である. 種々のガストリン代謝における研究より, 腎がガストリン破壊の主要部位であることが明らかにされた. ここで, 我々は, 免疫活性ガストリン (IRG) が, いわゆる “uremic gastropathy” に重要な役割を演じているかどうか調べるため, 2種類の抗血清-Rehfeldによってつくられた2604……Total IRG (total gastrin) を測定, 矢内原によるR2702……G34を測定-を使ったRIAによってIRGを測定した. 33名の慢性腎不全患者 (血液透析者24名, 非透析者9名) および12名の正常対照群において, 空腹時, 透析前後および食事負荷前後に採血を行った.
Shigeaki Muto, Naoki Murayama, Shigehiko Taki, Kenkichi Kato, Muneya Suzuki, Akito Ishibashi, Katsuji Takeda, Takao Oinuma, Norihiko Terao, Yasushi Asano, Saichi Hosoda, Michio Miyata   +11 more
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Helicobacterand Hypergastrinemia: The Quisling Option

Scandinavian Journal of Gastroenterology, 1991
Helicobacter infection of the gastric antrum is linked to the development of duodenal ulcer. A key element could be the associated hypergastrinemia. This brief commentary seeks answers to the following questions: 1) Does hypergastrinemia occur? 2) How does it occur? and 3) Does it matter?
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Hypergastrinemia in rheumatoid arthritis.

Clinical and experimental rheumatology, 1986
Fasting hypergastrinemia was noted in 13-41% of 82 of outpatients with definite or classical rheumatoid arthritis. No differences were noted between the normogastrinemic and the hypergastrinemic subjects in their age, sex, duration of rheumatoid disease, serological status, or medications.
A J, Yorke, P, Davis, M, Salkie, W, Weinstein, A B, Thomson   +4 more
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Ranitidine hydrochloride-induced hypergastrinemia.

Journal of the National Medical Association, 1985
Ranitidine hydrochloride (Zantac) is a recently released H(2)-receptor antagonist that is stated not to cause significant increases in serum gastrin. The case described herein demonstrates that this is not always true. Zantac produced significant acute hypergastrinemia, which was sustained for several days after the discontinuation of the drug.
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