Results 121 to 130 of about 56,321 (241)
THE SEPARATE EFFECTS OF HYPERPARATHYROIDISM, HYPERCALCEMIA OF MALIGNANCY, RENAL FAILURE, AND ACIDOSIS ON THE STATE OF CALCIUM, PHOSPHATE, AND OTHER IONS IN PLASMA* [PDF]
Mackenzie Walser
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Phosphate in Physiological and Pathological Mineralization: Important yet Often Unheeded
Phosphate serves as a building block for physiological mineralization, and as a signaling molecule that regulates the activity of mineralizing cells. The disturbance in these processes could induce a series of pathological mineralization, with abnormal mineralization of hard tissues and ectopic mineralization of soft tissues being the most ...
Wen Qin+8 more
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Tertiary Hyperparathyroidism [PDF]
D. R. Davies, C. E. Dent, L. Watson
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Plasma-chloride Levels in Hyperparathyroidism [PDF]
G. K. McGowan, M. R. Wills
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ABSTRACT Introduction Cardiovascular disease remains the primary cause of mortality after kidney transplantation. Hyperparathyroidism (HPT) may contribute to the accelerated progression of coronary artery disease (CAD) and peripheral artery disease (PAD), particularly in transplant patients.
Lane T. Cavey+7 more
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Renal Vein Thrombosis in Acute Hyperparathyroidism [PDF]
Ann‐Marie Pringle, E. K. M. Smith
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Molecular mechanism for transcriptional regulation of the parathyroid hormone gene by Epiprofin
The secretion of parathyroid hormone (PTH) from the chief cells in the parathyroid glands is regulated by the concentration of extracellular calcium. Epiprofin (Epfn) expression is induced by NFATc2 in response to elevated serum calcium, and Epfn negatively regulates PTH production.
Takashi Nakamura+8 more
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Hypo-hyperparathyroidism. [PDF]
Erica Allen, F J Millard, J. R. Nassim
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