Results 301 to 310 of about 174,025 (341)
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Oral Hypoglycemic Agents

Primary Care: Clinics in Office Practice, 1977
Oral agents should not be used in the treatment of patients with asymptomatic maturity-onset diabetes. The indication for sulfonylureas is symptomatic maturity-onset diabetes or excessive hyperglycemia--fasting blood sugar over 300 mg per 100 ml--in the elderly patient who cannot or will not take insulin. The use of biguanides cannot be recommended.
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Oral Hypoglycemic Agents

Archives of Internal Medicine, 1960
In early 1942 while investigating the effect of isopropylthiodiazole (2254 RP) in typhoid fever, M. Janbon and co-workers in the Infectious Disease Clinic at Montpellier Medical School in France found that this sulfanilamide derivative produced signs and symptoms resembling hypoglycemia.
W G, TOMHAVE, W J, KUHL
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Oral Hypoglycemic Agents

Primary Care: Clinics in Office Practice, 1988
This article reviews the history of oral hypoglycemic agents in the treatment of patients with non-insulin-dependent diabetes mellitus. It chronicles the rise and fall of the use of these agents as the major treatment modality for NIDDM, from their initial success in the late 1950's, to their being abandoned after the controversial findings of the ...
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Hypoglycemic Symptoms and the Hypoglycemic Experience

Psychosomatics, 1969
• The variety of the symptoms and the vagueness of the emotional distress which the pa­ tients with hypoglycemic episodes endure have long provided the physician with a baf­ fling battery of findings and complaints. In order to construct a useful foundation for helping patients in distress, it is necessary to have an organized and coherent under ...
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Hypoglycemic hemiplegic syndrome

Annals of Emergency Medicine, 1984
Hypoglycemia must be considered in any patient with an acute change of mental status or in a patient who shows a focal neurological deficit. Treatment with high IV glucose concentrations can save the patient from grave irreversible neurological sequelae and death.
R, Andrade   +8 more
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Hypoglycemic Brain Damage

Metabolic Brain Disease, 2004
Hypoglycemia was long considered to kill neurons by depriving them of glucose. We now know that hypoglycemia kills neurons actively from without, rather than by starvation from within. Hypoglycemia only causes neuronal death when the EEG becomes flat. This usually occurs after glucose levels have fallen below 1 mM (18 mg/dl) for some period, depending ...
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Potential Hypoglycemic Sulfonylureas

Journal of Pharmaceutical Sciences, 1964
A series of N ′-alkyl- N - p -phenylbenzenesulfonylureas and N , N ′-bis ( N -alkylcarbamyl) 4,4′-biphenyldisulfonamides has been prepared for pharmacological studies of their hypoglycemic activities.
K A, NIEFORTH, G L, JENKINS, A M, KNEVEL
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Hypoglycemic brain injury

Seminars in Neonatology, 2001
Hypoglycemia frequently occurs in newborn infants who previously have suffered asphyxia, who are offspring of diabetic mothers, or who are low birthweight for gestational age (IUGR). Many infants who are hypoglycemic do not exhibit clinical manifestations, while others are symptomatic and at risk for the occurrence of permanent brain damage.
R C, Vannucci, S J, Vannucci
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Oral Hypoglycemic Agents

New England Journal of Medicine, 1989
ORAL hypoglycemic agents are commonly prescribed drugs. In the United States they account for about 1 percent of all prescriptions.1 This review discusses the pharmacology, mechanisms of action, ef...
John A. Oates   +2 more
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Oral Hypoglycemic Agents

Pediatrics, 1969
I noted with interest the article by Drs. Adam and Schwartz, "Should Oral Hypoglycemic Agents Be Used in Pediatric and Pregnant Patients?" (Pediatrics, 42:819, 1968). In regard to juvenile diabetics, his conclusions were an echo of what usually appears in the ped iatric literature, namely, these drugs have no place in the management of any pediatric ...
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