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Hypophosphatemia and Hyperphosphatemia
Critical Care Clinics, 1991Pathophysiology, clinical sequelae, and treatment for hypophosphatemia and hyperphosphatemia are discussed. Hypophosphatemia results from a variety of conditions including malnutrition, carbohydrate refeeding, acid-base disorders, and hormonal and drug effects. Patients suffering from severe hypophosphatemia may present with a variety of syndromes that
Tusar K. Desai+2 more
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Approach to Treatment of Hypophosphatemia
American Journal of Kidney Diseases, 2012Hypophosphatemia can be acute or chronic. Acute hypophosphatemia with phosphate depletion is common in the hospital setting and results in significant morbidity and mortality. Chronic hypophosphatemia, often associated with genetic or acquired renal phosphate-wasting disorders, usually produces abnormal growth and rickets in children and osteomalacia ...
Arnold J. Felsenfeld, Barton S. Levine
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Hypophosphatemia and Reyeʼs syndrome
Critical Care Medicine, 1985Hypophosphatemia may occur in Reye's syndrome. We retrospectively studied 42 patients with Reye's syndrome to determine the frequency and degree of hypophosphatemia, to identify possible causes, and to determine whether hypophosphatemia was related to the severity of disease or mortality.
Robert K. Kanter, John L. Carroll
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Hypophosphatemia and Calcium Nephrolithiasis
Nephron Experimental Nephrology, 2004Our knowledge of phosphate balance under physiological and pathological situations has increased substantially during the last decade thanks to the molecular identification of three dissimilar families of sodium-phosphate cotransport systems, two of them almost exclusively expressed in epithelia whereas the third one has a ubiquitous expression ...
Dominique Prié+3 more
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Hypophosphatemia-induced Cardiomyopathy
The American Journal of the Medical Sciences, 2016Relatively few studies have been conducted to evaluate the effect of hypophosphatemia on cardiac function. The goal of this review was to determine whether there is an association between hypophosphatemia and cardiac function and to increase awareness of hypophosphatemia-induced cardiomyopathy as a new clinical entity and a reversible cause of heart ...
Sari Umekawa+4 more
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Phosphorus Deficiency and Hypophosphatemia
Hospital Practice, 1977Low serum phosphorus levels, sometimes associated with depletion of phosphorus stores, can engender a variety of serious, often life-threatening physiologic changes. The proximate cause of this dangerous situation is usually medical intervention in such conditions as alcoholism and diabetic ketoacidosis, which can produce a shift of phosphorus within ...
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Cardiovascular consequences of hypophosphatemia
Panminerva Medica, 2017Few studies have been conducted to evaluate the effect of hypophosphatemia on cardiovascular consequences. The goal of this review was to determine whether hypophosphatemia is associated with cardiovascular consequences and to increase its awareness as a new clinical entity and a reversible cause of cardiovascular consequences.We searched MEDLINE and ...
Nobuhiro Ariyoshi+4 more
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Assessment of Hyperphosphatemia and Hypophosphatemia
Clinics in Laboratory Medicine, 1993Methodologic aspects including causes of factitious hyperphosphatemia and hypophosphatemia are summarized. The differential diagnosis of hyperphosphatemia is reviewed under its three broad causes: decreased glomerular filtration rate, increased exogenous or endogenous phosphate load, and increased renal tubular phosphate reabsorption.
Bourke E, Yanagawa N
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The Psychiatric Aspect of Hypophosphatemia [PDF]
A patient who developed a psychotic state in association with hypophosphatemia is presented. There was no evidence for other causes of psychosis and the clinical features disappeared with phosphate replacement. It is suggested that hypophosphatemia be considered in a patient with an unexplained acute psychosis.
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Hypophosphatemia and metabolic acidosis.
Minerva anestesiologica, 2005The aim of the paper was to describe an unusual case of non lactic metabolic acidosis connected to hypophosphatemia and refractory to infusion of bicarbonate. A 37 year old man was admitted to Intensive Care Unit with a severe metabolic acidosis. On admission the arterial gas analysis showed non lactic metabolic acidosis (pH 7.17; base excess [BE] -20 ...
PALMESE S, PEZZA M, DE ROBERTIS E
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