Results 51 to 60 of about 322,048 (294)

Ictal epileptic headache as "subtle" symptom in generalized idiopathic epilepsy

open access: yes, 2012
Epilepsy and migraine are common neurologic chronic disorders with episodic manifestations characterized by recurrent attacks and a return to baseline conditions between attacks.
LAPENTA, Leonardo   +9 more
core   +1 more source

Global, regional, and national burden of epilepsy, 1990–2021: a systematic analysis for the Global Burden of Disease Study in 2021

open access: yesCost Effectiveness and Resource Allocation
Background Idiopathic epilepsy is a serious neurological disorder that can lead to premature death and severe disability. We aimed to assess the global burden of idiopathic epilepsy, to provide a comprehensive understanding of the current dynamics and ...
Tianqi Sun, Tianfu Yu, Pengcheng Liu
doaj   +1 more source

Migreine and periictal headache in patients with epilepsy [PDF]

open access: yesСаратовский научно-медицинский журнал, 2016
Migraine and epilepsy are diseases characterized by paroxysmal occurring clinical manifestations that have different mechanisms of pathogenesis. In the interictal period in 59% of patients with epilepsy there are different types of primary headache: 18%,
Muzalevskaia D.S.   +2 more
doaj  

(NEW) Electrocardiographic Changes In Children With Idiopathic Epilepsy And Epilepsy Secondary To Structural Brain Lesions [PDF]

open access: yesMinia Journal of Medical Research
Background:  Several changes in autonomic functions were associated with epileptic seizures, which may contribute immediately or soon after the ictal event to cardiovascular, pulmonary, gastrointestinal, and urinary manifestations.
Marwa Ali   +3 more
doaj   +1 more source

Genetic analysis of human absence epilepsy [PDF]

open access: yes, 2010
Idiopathic Mendelian epilepsies have been typically identified as channelopathies. Evidence suggests that mutations in genes encoding GABAA receptors, GABAB receptors or voltage-dependent calcium channels (VDCCs) may underlie childhood absence epilepsy ...
Robinson, R.A.
core  

Impact of Age on the Diagnostic Yield of Routine EEG in People With Childhood or Juvenile Absence Epilepsy: A Cross‐Sectional Study

open access: yesAnnals of Clinical and Translational Neurology, EarlyView.
ABSTRACT Background We aimed to identify the proportion of individuals with a confirmed diagnosis of childhood absence epilepsy (CAE) or juvenile absence epilepsy (JAE) who show a negative routine EEG (rEEG), and to determine the main factors associated with this finding.
Francesco Fortunato   +7 more
wiley   +1 more source

Idiopathic epilepsy and school achievement. [PDF]

open access: yesArchives of Disease in Childhood, 1994
Forty one children (20 boys, 21 girls) aged 6-10.8 years (mean age 8.6 years) who were affected with idiopathic epilepsy underwent neuropsychological (Wechsler Intelligence Scale for Children, Bender test) and behavioural assessment (Personality Inventory for Children; this was also used in a matched control group).
STURNIOLO, Maria Gabriella   +1 more
openaire   +3 more sources

Single‐Cell Profiling Reveals Clonally Expanded CX3CR1+ T Cells in Anti‐NMDA Receptor Encephalitis

open access: yesAdvanced Science, EarlyView.
CX3CR1+ T cells are associated with peripheral and central immune alterations in anti‐NMDA receptor encephalitis (NMDAR‐E). They show potential responsiveness to GluN1 (NR1) peptides, potential interactions with peripheral B cells, clonal expansion, increased CD137/CD154 expression, and inflammatory cytokine production. Their preferential cerebrospinal
Lin Yan   +13 more
wiley   +1 more source

The ability of executive functions to predict deterioration in social abstraction in adolescents with idiopathic generalized epilepsy

open access: yesMiddle East Current Psychiatry, 2022
Background Epilepsy is a prevalent disease worldwide. Executive functions and social cognition are essential in daily living functioning, academic performance, and social adjustment.
Saber Hadad   +4 more
doaj   +1 more source

Drug Repurposing for Treating NCOR‐Related Intellectual Disability

open access: yesAdvanced Science, EarlyView.
An autism‐associated NCOR1 heterozygous mutation upregulates NRGs and activates NRG backward signaling in hypothalamic GABAergic neurons in mouse models, leading to Gabra2 downregulation and cognitive deficits, which can be therapeutically targeted by repurposing FDA‐approved drugs.
Yan Kong   +15 more
wiley   +1 more source

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