Results 141 to 150 of about 737 (163)

A Change of Hallmark: An Update. [PDF]

open access: yesCells
Donnem T   +3 more
europepmc   +1 more source

Phosphoproteomic Profiling Reveals mTOR Signaling in Sustaining Macrophage Phagocytosis of Cancer Cells. [PDF]

open access: yesCancers (Basel)
Wang B   +8 more
europepmc   +1 more source

A Comprehensive Review of Current Advancements in the Diagnosis, Treatment, and Long-Term Implications of Hypertensive Disorders of Pregnancy. [PDF]

open access: yesCureus
Fukuya FA   +10 more
europepmc   +1 more source

Application of intervention mapping in supportive periodontal therapy of chronic periodontitis patients

open access: yesHuli yanjiu
ZHANG Jimin   +7 more
doaj  

Tocotrienols reverse IKAP and monoamine oxidase deficiencies in familial dysautonomia

Biochemical and Biophysical Research Communications, 2005
Familial dysautonomia (FD), a recessive neurodegenerative disease, is caused by mutations in the IKBKAP gene that result in the production of nonfunctional IKAP protein. Manifestations of FD include autonomic crises characterized by hypertension, tachycardia, diaphoresis, and vomiting. Elevated plasma levels of norepinephrine (NE) and dopamine observed
Sylvia L, Anderson, Berish Y, Rubin
exaly   +3 more sources

EGCG corrects aberrant splicing of IKAP mRNA in cells from patients with familial dysautonomia

Biochemical and Biophysical Research Communications, 2003
Familial dysautonomia (FD) is an autosomal recessive neurodegenerative disorder. The most prevalent causative mutation is a T-->C transition in a donor splice site of the IKBKAP transcript, resulting in aberrant splicing and a truncated protein. The mutation's position and leaky nature suggested that its impact might be moderated by altering the level ...
Sylvia L, Anderson   +2 more
exaly   +3 more sources

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