Results 211 to 220 of about 3,195,227 (346)

Immune cells dying from ferroptosis: mechanisms and therapeutic opportunities. [PDF]

open access: yesCell Death Dis
Liu J   +7 more
europepmc   +1 more source

Wnt Signaling in the Regulation of Immune Cell and Cancer Therapeutics [PDF]

open access: gold, 2019
Muhammad Haseeb   +3 more
openalex   +1 more source

Diabetes‐induced vascular calcification is associated with low pyrophosphate and its oral supplementation prevents calcification in diabetic mice

open access: yesFEBS Open Bio, EarlyView.
Induction of diabetes in three different mouse strains uniformly resulted in an increase in TNAP activity and a reduction in pyrophosphate (PPi) in the circulation. Inhibition of TNAP restored plasma PPi. Diabetes‐induced calcification in the media layer of the aorta was detected only in the Abcc6−/− strain, which is predisposed to ectopic ...
Krisztina Fülöp   +13 more
wiley   +1 more source

Alterations in Resident Immune Cells in Prenatal Trisomy 21 Lungs. [PDF]

open access: yesCells
Frauenpreis A   +9 more
europepmc   +1 more source

CD34+ Stem Cell Selection and CD3+ Addback for Pediatric Recipients of Matched Unrelated Adult Donor (MUD) Peripheral Blood Stem Cell Transplantation (PBSCT) Preliminary Results of Day 100 Trm, Immune Reconstitution (IR), PTLD, Systemic Viral Infections (SVI), and Invasive Fungal Infections (IFI)

open access: hybrid, 2011
Angela Ricci   +17 more
openalex   +1 more source

Establishment of a coculture system for Porphyromonas gingivalis and head and neck squamous cell carcinoma using spheroid culture and LATS inhibition

open access: yesFEBS Open Bio, EarlyView.
We established a spheroid coculture system enabling viable Porphyromonas gingivalis–HNSCC interactions under normoxic conditions. Inhibition of LATS1/2 maintains tumor cells in an undifferentiated state, which may promote spheroid growth and create a more permissive environment for bacterial persistence.
Yurika Nakajima   +4 more
wiley   +1 more source

DDX3X induces mesenchymal transition of endothelial cells by disrupting BMPR2 signaling

open access: yesFEBS Open Bio, EarlyView.
Elevated DDX3X expression led to downregulation of BMPR2, a key regulator of endothelial homeostasis and function. Our co‐immunoprecipitation assays further demonstrated a molecular interaction between DDX3X and BMPR2. Notably, DDX3X promoted lysosomal degradation of BMPR2, thereby impairing its downstream signaling and facilitating endothelial‐to ...
Yu Zhang   +7 more
wiley   +1 more source

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