Results 131 to 140 of about 189,422 (317)

Evaluation of Entropy Analysis as a Fault-Related Feature for Detecting Faults in Induction Motors and Their Kinematic Chain [PDF]

open access: gold
Arturo Yosimar Jaen-Cuéllar   +3 more
openalex   +1 more source

Intein‐based modular chimeric antigen receptor platform for specific CD19/CD20 co‐targeting

open access: yesMolecular Oncology, EarlyView.
CARtein is a modular CAR platform that uses split inteins to splice antigen‐recognition modules onto a universal signaling backbone, enabling precise, scarless assembly without re‐engineering signaling domains. Deployed here against CD19 and CD20 in B‐cell malignancies, the design supports flexible multi‐antigen targeting to boost T‐cell activation and
Pablo Gonzalez‐Garcia   +9 more
wiley   +1 more source

Triaxial bearing vibration dataset of induction motor under varying load conditions. [PDF]

open access: yesData Brief, 2022
Kumar D   +5 more
europepmc   +1 more source

Cytoplasmic p21 promotes stemness of colon cancer cells via activation of the NFκB pathway

open access: yesMolecular Oncology, EarlyView.
Cytoplasmic p21 promotes colorectal cancer stem cell (CSC) features by destabilizing the NFκB–IκB complex, activating NFκB signaling, and upregulating BCL‐xL and COX2. In contrast to nuclear p21, cytoplasmic p21 enhances spheroid formation and stemness transcription factor CD133.
Arnatchai Maiuthed   +10 more
wiley   +1 more source

A Method of Speed-sensorless Control of Induction Motor Based on MRAS

open access: yesGong-kuang zidonghua, 2012
In view of problem that traditional speed identification algorithm of induction motor only indentifies motor speed but does not consider influence of change of stator resistance on identification of motor speed, the paper proposed an improved method of ...
WANG Hong-yu, YE Hao, HOU Yan
doaj  

Induction Motor

open access: yesInduction Motor
種別 ...
openaire  

Class IIa HDACs forced degradation allows resensitization of oxaliplatin‐resistant FBXW7‐mutated colorectal cancer

open access: yesMolecular Oncology, EarlyView.
HDAC4 is degraded by the E3 ligase FBXW7. In colorectal cancer, FBXW7 mutations prevent HDAC4 degradation, leading to oxaliplatin resistance. Forced degradation of HDAC4 using a PROTAC compound restores drug sensitivity by resetting the super‐enhancer landscape, reprogramming the epigenetic state of FBXW7‐mutated cells to resemble oxaliplatin ...
Vanessa Tolotto   +13 more
wiley   +1 more source

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