Global remodeling of ADP-ribosylation by PARP1 suppresses influenza A virus infection. [PDF]
Zhang Z +17 more
europepmc +1 more source
Friend or Foe: The Role of the Cytoskeleton in Influenza A Virus Assembly [PDF]
Sukhmani Bedi, Akira Ono
openalex +1 more source
LMO7 Suppresses Tumor‐Associated Macrophage Phagocytosis of Tumor Cells Through Degradation of LRP1
LMO7 in tumor‐associated macrophages suppresses phagocytosis of tumor cells and limits cytotoxic T lymphocytes infiltration, fostering tumor progression. Mechanistically, LMO7 mediates the ubiquitination and degradation of the phagocytic receptor LRP1, impairing its ability to engulf tumor cells and driving macrophages toward an antitumor phenotype ...
Mengkai Li +12 more
wiley +1 more source
The cellular activating protein-1 cFos regulates influenza A virus replication. [PDF]
Gerodez A +7 more
europepmc +1 more source
Inactivation of Human Influenza Virus Using Gamma Irradiation
Ailar Sabbaghi +4 more
openalex +1 more source
EXPERIMENTAL TRANSMISSION OF INFLUENZA VIRUS INFECTION IN MICE [PDF]
Jerome L. Schulman
openalex +1 more source
XIAP Stabilizes DDRGK1 to Promote ER‐Phagy and Protects Against Noise‐Induced Hearing Loss
Mechanism of GAS‐mediated protection against noise‐induced hearing loss (NIHL). Noise exposure activates the ATF4/eIF2α axis, downregulating XIAP and promoting DDRGK1 degradation, thereby inhibiting ER‐phagy and leading to hair cell (HC) death. GAS treatment rescues XIAP and DDRGK1 expression, reactivating ER‐phagy to mitigate HC loss, synaptic damage,
Lin Yan +13 more
wiley +1 more source
The ribosomal landscape in influenza A virus infection: from molecular mechanisms to clinical relevance. [PDF]
Pott J, Kryvenko V, Vadász I.
europepmc +1 more source
An H5N1-based matrix protein 2 ectodomain tetrameric peptide vaccine provides cross-protection against lethal infection with H7N9 influenza virus [PDF]
Ho-Chuen Leung +7 more
openalex +1 more source
Nuclear Factor I‐B Delays Liver Fibrosis by Inhibiting Chemokine Ligand 5 Transcription
This study identifies the transcription factor Nuclear Factor I‐B (NFIB) as a key suppressor of liver fibrosis. NFIB expression declines during hepatic stellate cell activation, and its overexpression reduces fibrosis in mice models. The mechanism involves NFIB directly repressing chemokine C─C motif ligand 5 (CCL5), thereby alleviating oxidative ...
Qianqian Chen +14 more
wiley +1 more source

