Das liest die LIBREAS, Nummer #6 (Winter / Frühling 2020)
Redaktion Libreas
doaj +1 more source
Factors affecting time to publication in information science. [PDF]
Taşkın Z+3 more
europepmc +1 more source
Dual targeting of AKT and mTOR using MK2206 and RAD001 reduces tumor burden in an intracardiac colon cancer circulating tumor cell xenotransplantation model. Analysis of AKT isoform‐specific knockdowns in CTC‐MCC‐41 reveals differentially regulated proteins and phospho‐proteins by liquid chromatography coupled mass spectrometry. Circulating tumor cells
Daniel J. Smit+19 more
wiley +1 more source
Accelerating ethics, empathy, and equity in geographic information science. [PDF]
Nelson TA, Goodchild MF, Wright DJ.
europepmc +1 more source
Inhibitor of DNA binding‐1 is a key regulator of cancer cell vasculogenic mimicry
Elevated expression of transcriptional regulator inhibitor of DNA binding 1 (ID1) promoted cancer cell‐mediated vasculogenic mimicry (VM) through regulation of pro‐angiogenic and pro‐cancerous genes (e.g. VE‐cadherin (CDH5), TIE2, MMP9, DKK1). Higher ID1 expression also increased metastases to the lung and the liver.
Emma J. Thompson+11 more
wiley +1 more source
Three coordinate systems for information science approaches [PDF]
Tosio Kitagawa
openalex +2 more sources
Bohr: Physique atomique et connaissance humaine/Becker: Theorie der Elektrizität Bd. I/Schaefer und Päsler: Einführung in die Theoret. Physik Bd. I/MacDonald: Thermoelectricity: An Introduction to the Principles/Brillouin: Science and Information Theory/Bradley: Shock Waves in Chemistry and Physics/Pirani und Yarwood: Principles of Vacuum Engineering/Chase: Nuclear Pulse Spectrometry [PDF]
Klaus Pöschl+7 more
openalex +1 more source
Prediction of Research Hotspots Based on LSTM: Taking Information Science as Example. [PDF]
Xiang F.
europepmc +1 more source
Alectinib resistance in ALK+ NSCLC depends on treatment sequence and EML4‐ALK variants. Variant 1 exhibited off‐target resistance after first‐line treatment, while variant 3 and later lines favored on‐target mutations. Early resistance involved off‐target alterations, like MET and NF2, while on‐target mutations emerged with prolonged therapy.
Jie Hu+11 more
wiley +1 more source
This study used longitudinal transcriptomics and gene‐pattern classification to uncover patient‐specific mechanisms of chemotherapy resistance in breast cancer. Findings reveal preexisting drug‐tolerant states in primary tumors and diverse gene rewiring patterns across patients, converging on a few dysregulated functional modules. Despite receiving the
Maya Dadiani+14 more
wiley +1 more source