Results 141 to 150 of about 21,736,361 (314)

Minutes: Senate Committee on Information Technologies: April 2, 2013

, 2013
University of Minnesota: Senate Committee on Information Technologies. (2013). Minutes: Senate Committee on Information Technologies: April 2, 2013.
University of Minnesota: Senate Committee on Information Technologies
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On the Impact of Information Technologies Secondary-School Capacity in Business Development: Evidence From Smart Cities Around the World. [PDF]

Front Psychol, 2021
Barba-Sánchez V, Orozco-Barbosa L, Arias-Antúnez E.   +2 more
europepmc   +1 more source

Epigenetic silencing of the liver‐specific lncRNA LUNAR promotes liver cancer progression via NOTCH activation

Molecular Oncology, EarlyView.
LUNAR is a liver‐specific long noncoding RNA (lncRNA) that is highly expressed in normal liver but becomes epigenetically silenced in hepatocellular carcinoma through promoter hypermethylation. Loss of LUNAR is associated with NOTCH activation, epithelial–mesenchymal transition, and metastasis, whereas restoring LUNAR restrains metastatic progression ...
Se Ha Jang, Hyung Seok Kim, Geum Ok Baek, Moon Gyeong Yoon, Su In Lee, Jee‐Yeong Jeong, Ji Eun Han, Soon Sun Kim, Jae Youn Cheong, Jung Woo Eun   +9 more
wiley   +1 more source

Minutes: Senate Committee on Information Technologies: April 6, 2004

, 2004
UThink: Blogs at the University Libraries, Graduation Planner ProjectUniversity of Minnesota: Senate Committee on Information Technologies. (2004). Minutes: Senate Committee on Information Technologies: April 6, 2004.
University of Minnesota: Senate Committee on Information Technologies
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CEACAM1 participation in breast cancer progression

Molecular Oncology, EarlyView.
In invasive breast cancer (BC), CEACAM1 shifts from an apical to a uniform membranous/cytoplasmic pattern, or is lost, as tumors dedifferentiate, inversely tracking the Ki‐67 proliferative index. In MCF‐7 cells, only CEACAM1‐4L suppresses proliferation, repressing cell cycle and growth factor genes.
Mykola Lyndin, Irina Kube‐Golovin, Anatolii Romaniuk, Gunther Wennemuth   +3 more
wiley   +1 more source

Minutes: Senate Committee on Information Technologies: November 7, 2006

, 2006
NetFiles Update and Demonstration, Joint Classroom Advisory Committee/SCIT Meeting, OIT Budget RequestUniversity of Minnesota: Senate Committee on Information Technologies. (2006).
University of Minnesota: Senate Committee on Information Technologies
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Application of Information Technologies and Programming Methods of Embedded Systems for Complex Intellectual Analysis. [PDF]

Entropy (Basel), 2021
Emelianov V, Emelianova N, Zhilenkov A, Chernyi S.   +3 more
europepmc   +1 more source

Intrapatient tumour heterogeneity and clonal evolution in an autopsy study of metastatic salivary gland cancer

Molecular Oncology, EarlyView.
Tumour heterogeneity and clonal evolution of metastatic salivary gland cancer were evaluated in two patients with adenoid carcinoma and one patient with myoepithelial carcinoma. Radiology‐guided autopsy enabled multi‐region sampling (total samples n = 149), followed by whole‐genome sequencing and phylogenetic reconstruction (17 tumour samples, 4–7 per ...
Gerben Lassche, Niels J. van Ruitenbeek, Charlotte Adang, Luke O'Gorman, Pui Yuen Lee, Willemijn M. Klein, Adriana C. H. van Engen‐van Grunsven, Jack A. Schalken, Sander Bervoets, Gerald W. Verhaegh, Carla M. L. van Herpen   +10 more
wiley   +1 more source

Minutes: Senate Committee on Information Technologies: January 23, 2001

, 2001
Internet Guidelines Policy, Web Guidelines, Privacy Policy, VPNUniversity of Minnesota: Senate Committee on Information Technologies. (2001). Minutes: Senate Committee on Information Technologies: January 23, 2001.
University of Minnesota: Senate Committee on Information Technologies
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Somatostatin receptor 4 (SSTR4) is a tumor suppressor in cutaneous and head & neck squamous cell carcinomas

Molecular Oncology, EarlyView.
This study identifies somatostatin receptor 4 (Sstr4) as a critical tumor suppressor against skin and head/neck cancers (HNSCC, cSCC, and BCC). The loss of Sstr4 removes a check on cell growth, causing hyperactivation of the MAPK‐ERK signaling pathway (↑).
Ali Taqvi, Tuan Khang Huynh, Adway Kadam, Zahra Kardan, Yue Yan, Amina Jamal Laham, Sampath Kumar Loganathan   +6 more
wiley   +1 more source