Results 221 to 230 of about 656,131 (350)
Trajectory of Functional Decline Before and After Ischemic Stroke [PDF]
Mandip S. Dhamoon +4 more
openalex +1 more source
Brain Cell Senescence: A New Therapeutic Target for the Acute Treatment of Ischemic Stroke
Júlia Baixauli-Martín +7 more
openalex +1 more source
Chronic oral exposure to microplastics may disrupt gut microbiota homeostasis and intestinal barrier integrity, potentially engaging the gut–brain axis and systemic inflammatory responses. These alterations may be associated with impaired blood–brain barrier function, cerebral microvascular dysfunction, and enhanced endothelial inflammation, pro ...
Hongxing Wang +5 more
wiley +1 more source
The role of astrocytes in ischemic stroke - mechanisms, functions and treatment. [PDF]
Wang Y, Wang D, Li D, Ma Y.
europepmc +1 more source
A systematic review and meta-analysis of Ginsenoside-Rg1 (G-Rg1) in experimental ischemic stroke [PDF]
Chenglong Xie +5 more
openalex +1 more source
Cell‐Free DNA‐Based Theranostics for Inflammatory Disorders
Summary on the dual potential of cfDNA as biomarkers and therapeutic targets for inflammatory disorders. Figure was created with BioRender.com. ABSTRACT Inflammatory disorders are characterized by immune‐mediated inflammatory cascades that can affect multiple organs.
Jiatong Li +7 more
wiley +1 more source
Migrasomes in Ischemic Stroke: Molecular Landscape and Pathophysiological Impact
Multi‐omics profiling of stroke patients exposes systemic molecular fingerprints of immune activation, oxidative stress and metabolic collapse. Complement and coagulation cascade, and cholesterol metabolism emerge as shared functional hallmarks of plasma migrasomes in stroke patients and migrasomes isolated from experimental stroke mice.
Huifen Zhou +16 more
wiley +1 more source
Thrombolysis in an Ischemic Stroke Patient on Dabigatran Anticoagulation: A Case Report [PDF]
Luiz Carlos Porcello Marrone, A. Marrone
openalex +1 more source
PtRhIr/Ru SAN@M nanozymes cross the blood–brain barrier and selectively accumulate in hemorrhagic brain regions. By mimicking multiple enzyme activities, they attenuate oxidative stress, modulate microglial polarization toward an anti‐inflammatory phenotype, inhibit ferroptosis, and promote neuronal repair.
Jiebo Li +17 more
wiley +1 more source

