Results 61 to 70 of about 7,236 (206)
ChemPhysChem, Volume 27, Issue 9, 14 May 2026.The present study demonstrates the transformation of dual‐emissive H‐bonded molecular clusters of fluorophores into single emissive amorphous carbon nanodots and their structure‐property relationship in fluorescence‐based sensing of Bilirubin and Cu2+ ions. The present study aims to explore the origin of fluorescence in nitrogen‐doped carbon dots (NCDs)
Rajarshi Basu +6 more
wiley +1 more source
Haematalogical investigations in children [PDF]
, 2009 The haematology laboratory is able to perform a number of tests to help establish the cause of illness in children. The full blood count (FBC, also known as a complete blood count, CBC) is one of the most basic blood tests performed on children ...
Chalmers, E.A., Halsey, C.
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Neonatal hyperbilirubinemia: review of the current situation [PDF]
, 2011 OBJETIVO: identificar la incidencia de hiperbilirrubinemia neonatal. MÉTODO: estudio bibliométrico descriptivo transversal, realizado mediante búsquedas bibliográficas en la Biblioteca Virtual en Salud, en la Web of Science y en Medline con las palabras ...
Castaño Picó, María José +1 more
core +2 more sources
Health Science Reports, Volume 9, Issue 5, May 2026.ABSTRACT Background and Aims Crigler‐Najjar Syndrome (CNS) is a rare autosomal recessive disorder caused by uridine diphosphate glucuronosyltransferase (UGT1A1) deficiency, leading to unconjugated hyperbilirubinemia. Without treatment, patients are at high risk of kernicterus and irreversible neurological damage.
Sajad Teimoury +6 more
wiley +1 more source
Role of COHb Level in Newborns with ABO Blood Group Incompatibility in Predicting Newborn Jaundice Risk [PDF]
Forbes Tıp DergisiObjective: Neonates with hyperbilirubinemia are at risk of brain damage, and at least two-thirds of neonates show clinical signs of jaundice in the first week of life.
Kazım DARKA, Şahin TAKCI
doaj +1 more source
CRIGLER- NAJJAR SYNDROME – CASE REPORT
Slovenska pediatrija, 2022 Crigler-Najjar syndrome is a rare genetic disorder that causes severe unconjugated hyperbilirubinaemia. The syndrome is caused by a mutation in the UGT1A1 gene, which results in a deficiency or complete lack of the UGT1A1 enzyme, which is responsible for
Anja Šelih, Manca Velkavrh
doaj +1 more source
Veterinary Record Case Reports, Volume 14, Issue 2, May 2026.Abstract A 6‐year‐old spayed female Golden Retriever mix was diagnosed with immune‐mediated haemolytic anaemia. Initial treatments included immunosuppression and blood transfusions. With acute mentation decline and rapid increase in serum bilirubin, therapeutic plasma exchange (TPE) was initiated.
Natasha S. Yeh +2 more
wiley +1 more source
The return of metabolism: biochemistry and physiology of glycolysis
Biological Reviews, Volume 101, Issue 2, Page 751-803, April 2026.ABSTRACT Glycolysis is a fundamental metabolic pathway central to the bioenergetics and physiology of virtually all living organisms. In this comprehensive review, we explore the intricate biochemical principles and evolutionary origins of glycolytic pathways, from the classical Embden–Meyerhof–Parnas (EMP) pathway in humans to various prokaryotic and ...
Nana‐Maria Grüning +19 more
wiley +1 more source
Exchange Transfusion Trends and Risk Factors for Extreme Neonatal Hyperbilirubinemia over 10 Years in Shiraz, Iran [PDF]
Iranian Journal of Medical SciencesBackground: Exchange transfusion (ET) is an effective treatment for acute bilirubin encephalopathy and extreme neonatal hyperbilirubinemia (ENH). It can reduce mortality and morbidity.
Fariba Hemmati +4 more
doaj +1 more source
Progeny, December 2003, Vol. 19, no. 4 [PDF]
, 2003 This newsletter from The Department of Public Health about perinatal health care and ...
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