Results 151 to 160 of about 17,068 (196)
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Clinical Ketosis

Veterinary Clinics of North America: Food Animal Practice, 1988
A diagnosis of primary ketosis is based on clinical signs, clinical pathology, and ruling-out disorders that cause secondary ketosis. Various treatments can be used alone or in combination during the management of clinical ketosis. A treatment should be based on drugs with a mechanism of action that will eliminate the pathogenesis of the clinical signs
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POST-EXERCISE KETOSIS

The Lancet, 1969
Abstract The effects of ingestion of glucose after strenuous exercise on the concentrations of blood glucose, glycerol, and ketone-bodies, and plasma-free-fatty-acids (F.F.A.) have been studied in a group of twelve subjects. The results have been compared with those of oral glucose-tolerance tests at rest in eight of the same subjects and after ...
R H, Johnson   +3 more
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The Biochemical Lesion in Ketosis

Archives of Internal Medicine, 1961
Much information has come to light in recent years which bears on the problem of why ketone bodies accumulate in the mammalian body under certain conditions. I propose to discuss in this lecture observations which lead to the concept that ketosis is a disorder of the regulation of metabolic processes rather than an insufficiency of certain enzymes or ...
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Ketosis.

1940
An attempt has been made to present a comprehensive review of the different factors relating to the phenomenon of ketosis. Particular emphasis has been placed on the mechanism of the origin of acetone bodies, as well as on the site of their formation and utilization.
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THE TREATMENT OF KETOSIS IN LABOUR

BJOG: An International Journal of Obstetrics & Gynaecology, 1971
SummaryFifty patients who developed ketosis in labour were investigated and five different methods of treatment assessed. The clinical results and biochemical values were compared with those of a group of patients with non‐ketotic labours. It was concluded that few patients with ketonuria in labour are dehydrated, acidaemic or hypoglycaemic.
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On the biochemical etiology of ketosis

Life Sciences, 1964
Abstract When a low level of palmitate is added to a rat liver homogenate, more is converted to CO 2 than acetoacetate. However, as the level of palmitate is increased, the maximum capacity of the preferential pathway, namely complete oxidation to CO 2 , is reached and ketogenesis then predominates.
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Ketosis

Archives of Internal Medicine, 1961
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BOVINE KETOSIS

Nutrition Reviews, 2009
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Ruminant ketosis

Biochemical Society Transactions, 1981
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