Results 1 to 10 of about 33,008 (143)

Ribosomal protein L32 contributes to the growth, antibiotic resistance and virulence of Glaesserella parasuis. [PDF]

open access: yesFront Vet Sci
Glaesserella parasuis is the pathogen that causes Glässer’s disease in pigs, which is characterized by fibrinous polyserositis, arthritis and meningitis.
Chen Q   +8 more
europepmc   +6 more sources

Altered O-glycosylation of β<sub>1</sub>-adrenergic receptor N-terminal single-nucleotide variants modulates receptor processing and functional activity. [PDF]

open access: yesFEBS J
The human β1‐adrenergic receptor (β1AR) carries common (S49G) and rare (R31Q, A29T) single‐nucleotide polymorphisms (SNPs) in its extracellular N‐terminal domain. These were found to alter receptor O‐glycosylation and/or proteolytic cleavage, resulting in SNP‐dependent differences in β1AR cell surface expression and function that also involved the ...
Tuhkanen HE   +11 more
europepmc   +2 more sources

Muscle-specific Keap1 deletion enhances force production but does not prevent inactivity-induced muscle atrophy in mice. [PDF]

open access: yesFASEB J
In absence of oxidative stress (i.e., Control, left panel) Keap1 binds and ubiquitinates the antioxidant response element (ARE) transcription factor NRF2. Ubiquitinated NRF2 is degraded and unable to act as a transcription factor for ARE‐containing genes.
Miranda ER   +9 more
europepmc   +2 more sources

Ribosomal protein L32 enhances hepatocellular carcinoma progression

open access: yesCancer Medicine, 2023
AbstractPurposeThe underlying mechanisms of hepatocellular carcinoma (HCC) have not been fully investigated, and effective biomarkers for HCC are still needed to be explored. Therefore, our study sought to thoroughly examine the clinical significance and biological functions of the ribosomal protein L32 (RPL32) in HCC by coupling bioinformatic methods ...
Guoxin Hou   +3 more
openaire   +3 more sources

Chloroplast ribosomal protein L32 is encoded in the chloroplast genome [PDF]

open access: yesFEBS Letters, 1990
The 50 S subunit of chloroplast ribosomes was prepared from tobacco leaves. The proteins were fractionated and the N‐terminal amino acid sequence of a 14 kDa protein was determined. This sequence matches the N‐terminal sequence deduced from ORF55 located between ndhF and trnL on the small single‐copy region of tobacco chloroplast DNA.
Keita Torazawa   +5 more
openaire   +3 more sources

Identification of a polypeptide bound to the β region of the mouse r protein L32 promoter [PDF]

open access: yesFEBS Letters, 1991
Studies have been initiated to identify the protein component(s) which interact with the β regulatory region of the mouse ribosomal protein L32 gene promoter. By the combined use of the mobility shift assay and UV cross‐linking, a factor specific for the upstream transcriptional control sequence of the β region of the ribosomal protein L32 promoter has
Bruce H. Sells, Thillainathan Yoganathan
openaire   +3 more sources

Charged-Lepton Mixing and Lepton Flavor Violation [PDF]

open access: yes, 2015
We present a model for calculating charged-lepton mixing matrices. These matrices are an essential ingredient for predicting lepton flavor-violating rates in the lepton number nonuniversal models recently proposed to explain anomalies in B-meson decays ...
Guadagnoli, Diego, Lane, Kenneth
core   +8 more sources

Faint Population III supernovae as the origin of the most iron-poor stars [PDF]

open access: yes, 2014
The Astrophysical Journal Letters, 792:L32 (6pp), 2014 September 10. doi: 10.1088/2041-8205/792/2/L32. © 2014 The American Astronomical Society. All rights reserved.
Aoki   +27 more
core   +2 more sources

Lepton Flavor Violation in B Decays? [PDF]

open access: yes, 2014
The LHCb Collaboration's measurement of R_K = B(B+ -> K+ mu+ mu-)/B(B+ -> K+ e+e-) lies 2.6 sigma below the Standard Model prediction. Several groups suggest this deficit to result from new lepton non-universal interactions of muons.
Glashow, Sheldon L.   +2 more
core   +1 more source

Expression of A152T human tau causes age-dependent neuronal dysfunction and loss in transgenic mice. [PDF]

open access: yes, 2016
A152T-variant human tau (hTau-A152T) increases risk for tauopathies, including Alzheimer's disease. Comparing mice with regulatable expression of hTau-A152T or wild-type hTau (hTau-WT), we find age-dependent neuronal loss, cognitive impairments, and ...
Craft, Ryan   +13 more
core   +2 more sources

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