Results 121 to 130 of about 2,272 (172)
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Indian kala-azar caused by Leishmania tropica
Lancet, The, 1995Kala-azar, or visceral leishmaniasis, in India is generally assumed to be a result of infection with Leishmania donovani. 15 parasite isolates collected over the past 10 years from patients with classical disease were typed by monoclonal antibodies, isoenzymes, and kDNA analysis.
Franklin A Neva +2 more
exaly +3 more sources
Studies on the culture of Leishmania tropica
Transactions of the Royal Society of Tropical Medicine and Hygiene, 1939exaly +2 more sources
Leishmania tropica: Chemostatic cultivation
Experimental Parasitology, 1970Abstract A semidefined, cell-free liquid medium was developed which supported batch cultivation of L. tropica, L. donovani , and L. brasiliensis , after preadaptation over N.I.H. blood agar. Average batch growth peaks of 2.4 × 10 8 cells/ml for L. tropica , 4.1 × 10 7 cells/ml for L. donovani , and 9.9 × 10 7 cells/ml for L.
F W, Schaefer, E J, Bell, F J, Etges
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Characterization of Leishmania (Leishmania) tropica axenic amastigotes
Acta Tropica, 2010Optimum conditions for generating Leishmania (Leishmania) tropica axenic amastigotes (AxA) in culture were determined, pH 5.5/36 degrees C, and the parasites characterized by different techniques, including light microscopy, macrophage infection, stage specific antigen expression and differential display. AxA were morphologically similar to amastigotes
Abedelmajeed, Nasereddin +3 more
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Leishmania tropica and Leishmania mexicana: Cross-immunity in mice
Experimental Parasitology, 1978Abstract The effect of a previous or concurrent Leishmania tropica major infection on a L. mexicana infection was studied. Mice which were recovering from or had recovered from a L. tropica infection were found to be totally resistant to L. mexicana. Infection of mice already carrying a L. mexicana infection with L.
J, Alexander, R S, Phillips
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Mitochondrial proteome profiling of Leishmania tropica
Microbial Pathogenesis, 2019The mitochondrion of kinetoplastida has unique characteristics both in structure and function. To better understand the mitochondrial proteome of the Leishmania tropica promastigote stage, liquid chromatography coupled with mass spectrometry (LC/MS/MS) approach was used.
Minoo, Tasbihi +4 more
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Leishmania tropica Infections in Travellers
Archives of Dermatology, 1976• Oriental sore (acute cutaneous leishmaniasis) is usually a self-limited infection of the skin caused by the protozoan Leishmania tropica . This disease is endemic to the Mediterranean, Asia, Africa, and the Middle East, and is seen in this country among travellers and immigrants.
R C, Rau, H V, Dubin, W B, Taylor
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Thymoquinone Effect on Leishmania tropica/infantum and Leishmania-Infected Macrophages
Acta Parasitologica, 2023Leishmania is a parasitic protozoan that tries to enter and amplify within macrophages. Macrophage cells are also immune defense cells that phagocyte many microbes like bacteria, fungi, as well as parasites like Leishmania spp. However, they are unable to kill this parasite that resides in the phagosomes of contaminated macrophages and multiplies in ...
Amir KarimiPourSaryazdi +4 more
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The Metabolism of Leishmania Tropica
Journal of Infectious Diseases, 1928An extensive literature dealing with the metabolism of bacteria has accumulated, but so far, little work has been carried out on the metabolic activities of the protozoa. The protozoologist's inability to grow the latter for many generations, has been and still is one of the difficulties in the way.
A. J. Salle, C. L. A. Schmidt
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Archives of Dermatology, 1977
To the Editor.— The article on Leishmania tropica infections in travellers by Drs Rau, Dubin, and Taylor (112:197, 1976) was an excellent summary of the clinical and pathologic findings of this disease. However, I think that their conclusion not to treat this disease on the basis on four cases and a deficiency in the medical literature should not be ...
openaire +2 more sources
To the Editor.— The article on Leishmania tropica infections in travellers by Drs Rau, Dubin, and Taylor (112:197, 1976) was an excellent summary of the clinical and pathologic findings of this disease. However, I think that their conclusion not to treat this disease on the basis on four cases and a deficiency in the medical literature should not be ...
openaire +2 more sources