Results 151 to 160 of about 68,163 (199)
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Characterization of Leishmania (Leishmania) tropica axenic amastigotes
Acta Tropica, 2010Optimum conditions for generating Leishmania (Leishmania) tropica axenic amastigotes (AxA) in culture were determined, pH 5.5/36 degrees C, and the parasites characterized by different techniques, including light microscopy, macrophage infection, stage specific antigen expression and differential display. AxA were morphologically similar to amastigotes
Abedelmajeed, Nasereddin +3 more
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The Metabolism of Leishmania Tropica
Journal of Infectious Diseases, 1928An extensive literature dealing with the metabolism of bacteria has accumulated, but so far, little work has been carried out on the metabolic activities of the protozoa. The protozoologist's inability to grow the latter for many generations, has been and still is one of the difficulties in the way.
A. J. Salle, C. L. A. Schmidt
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Leishmania tropica and Leishmania mexicana: Cross-immunity in mice
Experimental Parasitology, 1978Abstract The effect of a previous or concurrent Leishmania tropica major infection on a L. mexicana infection was studied. Mice which were recovering from or had recovered from a L. tropica infection were found to be totally resistant to L. mexicana. Infection of mice already carrying a L. mexicana infection with L.
J, Alexander, R S, Phillips
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Mitochondrial proteome profiling of Leishmania tropica
Microbial Pathogenesis, 2019The mitochondrion of kinetoplastida has unique characteristics both in structure and function. To better understand the mitochondrial proteome of the Leishmania tropica promastigote stage, liquid chromatography coupled with mass spectrometry (LC/MS/MS) approach was used.
Minoo, Tasbihi +4 more
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Leishmania tropica Infections in Travellers
Archives of Dermatology, 1976• Oriental sore (acute cutaneous leishmaniasis) is usually a self-limited infection of the skin caused by the protozoan Leishmania tropica . This disease is endemic to the Mediterranean, Asia, Africa, and the Middle East, and is seen in this country among travellers and immigrants.
R C, Rau, H V, Dubin, W B, Taylor
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Thymoquinone Effect on Leishmania tropica/infantum and Leishmania-Infected Macrophages
Acta Parasitologica, 2023Leishmania is a parasitic protozoan that tries to enter and amplify within macrophages. Macrophage cells are also immune defense cells that phagocyte many microbes like bacteria, fungi, as well as parasites like Leishmania spp. However, they are unable to kill this parasite that resides in the phagosomes of contaminated macrophages and multiplies in ...
Amir KarimiPourSaryazdi +4 more
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Archives of Dermatology, 1977
To the Editor.— The article on Leishmania tropica infections in travellers by Drs Rau, Dubin, and Taylor (112:197, 1976) was an excellent summary of the clinical and pathologic findings of this disease. However, I think that their conclusion not to treat this disease on the basis on four cases and a deficiency in the medical literature should not be ...
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To the Editor.— The article on Leishmania tropica infections in travellers by Drs Rau, Dubin, and Taylor (112:197, 1976) was an excellent summary of the clinical and pathologic findings of this disease. However, I think that their conclusion not to treat this disease on the basis on four cases and a deficiency in the medical literature should not be ...
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Leishmania tropica: Uptake of methionine by promastigotes
Experimental Parasitology, 1977Abstract Promastigotes of Leishmania tropica transport methionine against a concentration gradient through a saturable system. This concentrative uptake requires metabolic energy and is sensitive to temperature and sulfhydryl reagents such as N -ethyl maleimide. Intracellular methionine exchanges with external methionine.
A J, Mukkada, M W, Simon
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Studies on the Culture of Leishmania tropica
Transactions of the Royal Society of Tropical Medicine and Hygiene, 1939exaly +2 more sources
Toxicity of bilirubin to Leishmania tropica promastigotes
Experimental Parasitology, 1976Abstract Treatment of Leishmania tropica promastigotes with bilirubin results in loss of viability and death of the cells. A concentration of 0.1 m M is sufficient to cause complete inhibition of growth. Bilirubin inhibits the uptake of 2-deoxy- d -glucose and l -methionine. It increases the efflux of intracellularly accumulated sugars (both free
M W, Simon, J M, Rusnak, A J, Mukkada
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