B‐cell chronic lymphocytic leukemia (B‐CLL) and monoclonal B‐cell lymphocytosis (MBL) show altered proteomes and phosphoproteomes, analyzed using mass spectrometry, protein microarrays, and western blotting. Identifying 2970 proteins and 316 phosphoproteins, including 55 novel phosphopeptides, we reveal BCR and NF‐kβ/STAT3 signaling in disease ...
Paula Díez+17 more
wiley +1 more source
NLRP3 inflammasome: a key player in the pathogenesis of life-style disorders. [PDF]
Ramachandran R, Manan A, Kim J, Choi S.
europepmc +1 more source
Epidemiological, life style, and occupational factors associated with lower limb varicose veins: a case control study. [PDF]
Elamrawy S+4 more
europepmc +1 more source
Effects of the Change of Life-style on Public Support and Car Use for Traffic Reduction Policy.
Nobuhiko Matsumura, Yasutsugu Nitta
openalex +2 more sources
Ubiquitination of transcription factors in cancer: unveiling therapeutic potential
In cancer, dysregulated ubiquitination of transcription factors contributes to the uncontrolled growth and survival characteristics of tumors. Tumor suppressors are degraded by aberrant ubiquitination, or oncogenic transcription factors gain stability through ubiquitination, thereby promoting tumorigenesis.
Dongha Kim, Hye Jin Nam, Sung Hee Baek
wiley +1 more source
Life-style and metabolic factors do not affect risk for glioma: a prospective population-based study (The Cohort of Norway). [PDF]
Gheorghiu A+5 more
europepmc +1 more source
Evolution of the fungal self-fertile reproductive life style from self-sterile ancestors
Sung‐Hwan Yun+3 more
openalex +1 more source
STEBBINS, Robert A., The Franco-Calgarians: French Language, Leisure and Linguistic Life-Style in an Anglophone City (Toronto, University of Toronto Press, 1994). [PDF]
Jean Pariseau
openalex +1 more source
THE DETERMINANTS OF CRIMINAL VICTIMIZATION IN SÃO PAULO STATE [PDF]
In this paper an exploratory research of the determinants of criminal victimization in São Paulo state is conducted using the life-style and opportunity models with Seade's 1998 Pesquisa de Condição de Vida (Life Condition Survey).
Fábio Augusto Reis Gomes+1 more
core
Nuclear prothymosin α inhibits epithelial‐mesenchymal transition (EMT) in lung cancer by increasing Smad7 acetylation and competing with Smad2 for binding to SNAI1, TWIST1, and ZEB1 promoters. In early‐stage cancer, ProT suppresses TGF‐β‐induced EMT, while its loss in the nucleus in late‐stage cancer leads to enhanced EMT and poor prognosis.
Liyun Chen+12 more
wiley +1 more source