Results 191 to 200 of about 8,390 (226)

Microtubule motors of opposite polarity cooperate rather than compete in cargo transport. [PDF]

Nat Struct Mol Biol
Markus SM.
europepmc   +1 more source

Structure and Function of Dynein's Non-Catalytic Subunits. [PDF]

Cells
Rao L, Gennerich A.
europepmc   +1 more source

Nde1 Promotes Lis1 Binding to Full-Length Autoinhibited Human Dynein-1

Jun Yang, Yuanchang Zhao, Pengxin Chai, Ahmet Yıldız, Kai Zhang   +4 more
openalex   +1 more source

Function of manchette and intra-manchette transport in spermatogenesis and male fertility. [PDF]

Cell Commun Signal
Gao T, Liu Y, Li J, Zhang Y, Wu B.
europepmc   +1 more source

LIS1-tiden ble til bok

Heidi Østvedt Strydom
openalex   +1 more source

Spatial Proteomics Reveals Distinct Protein Patterns in Cortical Migration Disorders Caused by LIN28A Overexpression and WNT Activation. [PDF]

Mol Cell Proteomics
Navolić J, Hawass S, Moritz M, Hahn J, Middelkamp M, Gocke A, Dottermusch M, Schumann Y, Ruck L, Krisp C, Godbole S, Sumislawski P, Köppen N, Gargioni E, Schlüter H, Neumann JE.   +15 more
europepmc   +1 more source

Lissencephaly with subcortical band heterotopia in an East African child: A case report. [PDF]

Radiol Case Rep
Ngowi E, Datoo A, Ally P, Salum H, Edward K.   +4 more
europepmc   +1 more source

COP9 signalosome and PRMT5 methylosome complexes are essential regulators of Lis1-dynein based transport

Devanshi Gupta, Subbareddy Maddika
openalex   +1 more source

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LIS1 functions in normal development and disease

Current Opinion in Neurobiology, 2013
LIS1, the first gene to be identified as involved in a neuronal migration disease, is a dosage-sensitive gene whose proper levels are required for multiple aspects of cortical development. Deletions in LIS1 result in a severe brain malformation, known as lissencephaly, whereas duplications delay brain development.
Orly Reiner, Tamar Sapir
exaly   +4 more sources