Results 151 to 160 of about 266,382 (322)

Autre cause de mort subite du nourrisson: à propos d’un cas clinique de syndrome du QT long congenital [PDF]

open access: yes, 2016
Le syndrome du QT long congénital est une maladie rythmique liée à une mutation génétique et caractérisée par un espace QT allongé sur l'électrocardiogramme, des arythmies malignes type torsade de pointe et  fibrillation ventriculaire entraînant une mort
Gobin, Eric   +2 more
core   +1 more source

Fascicular/Purkinje Tissue Colocalized With Scar in Cardiomyopathy Patients Undergoing Ventricular Fibrillation Ablation

open access: yesPacing and Clinical Electrophysiology, EarlyView.
ABSTRACT Background Ventricular fibrillation (VF) is a poorly understood arrhythmia that is one of the main mechanisms of sudden cardiac death in patients with structural heart disease (SHD). Fascicular and Purkinje tissue (FPT) has been implicated in VF.
Emir Baskovski   +8 more
wiley   +1 more source

Диагностика различных вариантов кардиомиопатий у детей. Новый взгляд на старую проблему (Часть 3) [PDF]

open access: yes, 2013
В статті наводиться власне спостереження сімейної форми аритмогенної дисплазії правого шлуночка у дівчинки, яка перебувала під спостереженням протягом 12 років.
Сухарева, Г.Э.
core  

Rational prediction of pharmacological treatment options for a novel KCNH2- linked variant of the Short QT Syndrome [PDF]

open access: yes
Congenital disorders of cardiac repolarization are associated with risk of serious arrhythmias and sudden death. The Long QT Syndrome (LQTS) is well-established to predispose towards torsades de pointes [1].
Jules C Hancox
core   +1 more source

Inhibitory effect of oestradiol on the cardiac KV7.1/KCNE1 channel is species dependent

open access: yesExperimental Physiology, EarlyView.
Abstract Oestradiol (17β‐E2) is reported to prolong the cardiac action potential duration and QT interval, in part by affecting cardiac ion channels. Previous studies found inhibiting 17β‐E2 effects on the repolarizating cardiac KV7.1/KCNE1 channel, or its native current, in heterologous expression systems or tissue from animal species.
Veronika A. Linhart   +2 more
wiley   +1 more source

Sudden Cardiac Arrest in the Postpartum Period Due to Long QT Syndrome and Dilated Cardiomyopathy

open access: yesJACC: Case Reports, 2023
We describe the case of a previously healthy patient presenting with sudden cardiac arrest in the postpartum period as a result of concomitant congenital type 1 long QT syndrome and BAG3 dilated cardiomyopathy.
Daniel R. Patterson, MD   +3 more
doaj  

The causative role of amyloidosis in the cardiac complications of Alzheimer's disease: a comprehensive systematic review

open access: yesThe Journal of Physiology, EarlyView.
Abstract figure legend Schematic illustration of the bidirectional causative link between cerebral amyloid‐beta (Aβ) angiopathy and cardiovascular disease in Alzheimer's disease (AD). Common cardiovascular risk factors like microvascular thrombosis, diabetes, atrial fibrillation, hypertension and atherosclerosis lead to cerebral hypoperfusion and ...
Samuel Parker   +2 more
wiley   +1 more source

In silico modelling of multi‐electrode arrays for enhancing cardiac drug testing on hiPSC‐CM heterogeneous tissues

open access: yesThe Journal of Physiology, EarlyView.
Abstract figure legend Schematic overview of the experimental and computational framework for investigating hiPSC‐CM electrophysiology with MEA systems. The MEA‐based model integrates experimental data with phenotype‐specific ionic models and tissue‐level heterogeneity.
Sofia Botti   +2 more
wiley   +1 more source

Prenatal diagnosis of long QT syndrome using fetal magnetocardiography [PDF]

open access: bronze, 1999
Hiromi Hamada   +7 more
openalex   +1 more source

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