Results 201 to 210 of about 86,066 (242)

Preliminary Results with Amrinone in Perioperative Low Cardiac Output Syndrome

The Thoracic and Cardiovascular Surgeon, 1987
The hemodynamic parameters of 24 cardiosurgical patients with low cardiac output syndrome (LCOS) after weaning from bypass were evaluated retrospectively. These patients were additionally treated with amrinone if the conventional therapy with catecholamines and vasodilators did not reach satisfying hemodynamic results.
M, Günnicker, W, Hess
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Inodilators in the Management of Low Cardiac Output Syndrome After Pediatric Cardiac Surgery

Current Vascular Pharmacology, 2015
Postoperative low cardiac output syndrome has been shown to have both a central and a peripheral vascular involvement. Therefore, inodilators which provide with a combination of positive inotropic and vasodilating therapy, conceptually should be an ideal form of treatment. However, contradictory data on these drugs exist.
Angela, Ferrer-Barba, Iria, Gonzalez-Rivera, Victor, Bautista-Hernandez   +2 more
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Pathogenesis of the low cardiac output syndrome in postresuscitation states

Bulletin of Experimental Biology and Medicine, 1976
Acute hypervolemia induced in experiments on dogs by infusion of dextran, did not produce decompensation of the circulation in animals whose cardiac output was sharply depressed in the postresuscitation period after circulatory arrest lasting 15 min.
I. E. Trubina, A. V. Volkov
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Intra-Aortic Balloon Pumping for Low Cardiac Output Syndromes

Surgical Clinics of North America, 1975
Since the prototype of the intra-aortic balloon pump was introduced, data accumulated from work with animals and in humans have begun to establish those clinical conditions amenable to partial temporary circulation support and to define the physiologic limits of this support system.
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[Perioperative Care of Low Cardiac Output Syndrome].

Kyobu geka. The Japanese journal of thoracic surgery, 2019
Low cardiac syndrome( LOS) is the imbalance of the oxygen supply and demand at the tissue due to reduced oxygen supply by decreased cardiac function. The causes of the cardiac dysfunction are insufficient cardioplegia, elongation of aortic clamp or cardiopulmonary bypass (CPB) duration, incomplete revascularization, and so on.
Hiroki, Kato, Yoshiro, Matsui
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Optimal Inotropic Support Strategy in Low Cardiac Output Syndrome

Seminars in Thoracic and Cardiovascular Surgery
Low cardiac output syndrome (LCOS), a form of cardiogenic shock that occurs after cardiac surgery, is associated with an elevated risk of morbidity and mortality. Generally, LCOS is managed medically with inotropes and vasopressors to optimize hemodynamics.
Travis J. Miles, Kyle W. Blackburn, Marc R. Moon, Subhasis Chatterjee   +3 more
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Treatment of Low Cardiac Output Syndrome-our experience.

Neurologia Croatica. Supplement, 1998
Predstavljamo naša iskustva u liječenju pacijenata sa sindromom niskog minutnog ...
Husedžinović Ino, Barišin Stjepan, Nikić Nada, Desnica Marina, Bošnjak Danica, Korušić Anđelko   +5 more
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Low-cardiac-output-Syndrom: Pathophysiologie und Behandlung

1983
Das sog. Low-cardiac-output-Syndrom stellt die kritischste Situation nach Herzoperationen dar. Das Bestreben des Herzchirurgen mus vor allem darauf gerichtet sein, das Auftreten dieser schwerwiegenden Komplikation durch grundliche Operationsvorbereitung, vorsichtige Narkosefuhrung, prazise Operationstechnik, konsequenten Myokardschutz und sorgfaltige ...
Bradley J. Harlan, Albert Starr
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Treatment of low cardiac output syndrome with phosphodiesterase inhibitors

Acta medica Croatica. Supplement, 1997
liječenje sindroma niskog minutnog volumena inhibitorima ...
Husedžinović Ino, Korušić Anđelko, Tonković Dinko, Desnica Marina, Bošnjak Danica   +4 more
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[Pathogenesis of the low cardiac output syndrome in postreanimation states].

Biulleten' eksperimental'noi biologii i meditsiny, 1976
Functional reserves of the cardiovascular system during sharp depression of the cardiac output in the postreanimation period after 15-minute cardiac arrest were studied in experiments on dogs (by loading with different fluid volumes). Acute hypervolemia did not produce any circulatory decompensation.
I E, Trubina, A V, Volkov
openaire   +1 more source