Results 161 to 170 of about 946,279 (310)
Aspects of Ω-deformed M-theory
We explore the properties of Ω-deformed M-theory, with particular focus on the ℂ ϵ 1 $$ {\mathbb{C}}_{\epsilon_1} $$ × ℂ ϵ 2 $$ {\mathbb{C}}_{\epsilon_2} $$ × ℂ ϵ 3 $$ {\mathbb{C}}_{\epsilon_3} $$ background and coupling to Ω-deformed M2 and M5 brane ...
Davide Gaiotto, Jihwan Oh
doaj +1 more source
Emerging role of ARHGAP29 in melanoma cell phenotype switching
This study gives first insights into the role of ARHGAP29 in malignant melanoma. ARHGAP29 was revealed to be connected to tumor cell plasticity, promoting a mesenchymal‐like, invasive phenotype and driving tumor progression. Further, it modulates cell spreading by influencing RhoA/ROCK signaling and affects SMAD2 activity. Rho GTPase‐activating protein
Beatrice Charlotte Tröster +3 more
wiley +1 more source
Duality Symmetric String and M-Theory [PDF]
David S. Berman, Daniel C. Thompson
openalex +1 more source
Following high dose rate brachytherapy (HDR‐BT) for hepatocellular carcinoma (HCC), patients were classified as responders and nonresponders. Post‐therapy serum induced increased BrdU incorporation and Cyclin E expression of Huh7 and HepG2 cells in nonresponders, but decreased levels in responders.
Lukas Salvermoser +14 more
wiley +1 more source
Scalar Cosmological Perturbations in M-theory with Higher Derivative Corrections [PDF]
Kazuho Hiraga, Yoshifumi Hyakutake
openalex +1 more source
A synthetic benzoxazine dimer derivative targets c‐Myc to inhibit colorectal cancer progression
Benzoxazine dimer derivatives bind to the bHLH‐LZ region of c‐Myc, disrupting c‐Myc/MAX complexes, which are evaluated from SAR analysis. This increases ubiquitination and reduces cellular c‐Myc. Impairing DNA repair mechanisms is shown through proteomic analysis.
Nicharat Sriratanasak +8 more
wiley +1 more source
Theory and practice of direct methods in crystallographyedited by M. F. C. Ladd and R. A. Palmer [PDF]
J. H. Robertson
openalex +1 more source
Adaptaquin selectively kills glioma stem cells while sparing differentiated brain cells. Transcriptomic and proteomic analyses show Adaptaquin disrupts iron and cholesterol homeostasis, with iron chelation amplifying cytotoxicity via cholesterol depletion, mitochondrial dysfunction, and elevated reactive oxygen species.
Adrien M. Vaquié +16 more
wiley +1 more source

