Results 121 to 130 of about 1,293 (164)
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Immune response to maedi-visna virus

Frontiers in Bioscience, 2007
The ovine maedi-visna virus (MVV) was the first lentivirus to be isolated and characterized 1957 in Iceland. MVV leads to a life-long, persistent infection with slow development of lesions in the lung and the central nervous system (CNS). The main target cells of MVV are of the monocyte/macrophage lineage and it does not infect T-lymphocytes or cause ...
H. Arnarson   +3 more
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Ultrastructural studies on Maedi-Visna virus

Archives of Virology, 1980
Ultrastructural studies of Maedi-Visna virus (MVV) particles isolated from tissue culture fluids of MVV-infected cells as well as cultured cells infected with MVV were performed. MVV particles aree bounded by an envelope with projections loosely attached to its surface.
M. Bruns, F. Weiland
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Maedi/visna: a review

Veterinary Record, 1980
A brief history of the occurrence of maedi/visna or maedi/visna related diseases is given. The clinical features and diagnosis are described. Recent experimental work performed in an attempt to resolve the pathogenetic features of slowness and persistence of infection, characteristic of maedi/visna, is covered.
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Concern over maedi visna breakdowns

Veterinary Record, 2014
WE would like to raise our concerns with veterinary colleagues over an apparent increase in the number of flocks in the maedi visna (MV) accreditation scheme that have had a breakdown with MV infection. Most of the blood testing for the MV accreditation scheme takes place during the summer months; however, so far this year there have been nine ...
Catriona Ritchie, Brian Hosie
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Changes to the SAC's maedi‐visna accreditation scheme [PDF]

open access: possibleVeterinary Record, 2008
SIR, — The Scottish Agricultural College (sac) has examined the maedi-visna (mv) accreditation scheme, together with industry representatives, in an effort to make it less expensive in view of the severe financial pressures faced by the sheep industry.
Ian Pritchard, Brian Hosie, Barti Synge
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Maedi-Visna and Related Diseases

1989
1. Introduction.- 2. Maedi-Visna. History and Clinical Description.- 3. Maedi-Visna. Pathology and Pathogenesis.- 4. Maedi-Visna. Etiology and Immune Response.- 5. Diagnostic Methods.- 6. Economie Importance, Epidemiology and Control.- 7. Pathology and Epidemiology of Lentiviral Infection of Goats.- 8.
G. Pétursson, R. Hoff-Jørgensen
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The biology of maedi-visna virus—An overview

British Veterinary Journal, 1993
This review aims to summarize the current understanding of the biology of maedi-visna virus (MVV), the prototype virus of the family lentivirinae. The paper provides a short overview of the historical background to the discovery of MVV. Detailed descriptions of the structure and organization of the MVV genome and of the virion encoded polypeptides are ...
Robert G. Dalziel, Nessa Carey
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Studies in epidemiology of maedi/visna in sheep

Research in Veterinary Science, 1979
Lambs born to ewes from flocks with a high incidence of maedi/visna were separated from their dams at birth (group 1), or after 10 h (group 2), six weeks (group 3) or one year (group 4) and were observed for periods of up to eight years. Group 1 lambs remained free of infection while 28 per cent, 76 per cent and 81 per cent respectively of lambs in the
G.F. De Boer   +3 more
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Maedi-Visna and Ovine Progressive Pneumonia

Veterinary Clinics of North America: Food Animal Practice, 1997
Maedi-Visna and ovine progressive pneumonia are disease of sheep that are caused by ovine lentivirus and characterized by chronic inflammation of the lungs, mammary glands, joints, and central nervous system. Although tremendous progress in research has led to a better understanding of the pathogenesis of these diseases, many questions still remain ...
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Cross Neutralization of Maedi‐Visna Virus Strains*

Zentralblatt für Veterinärmedizin Reihe B, 1984
SummaryCross‐neutralization of four Norwegian, two Icelandic and one Danish maedi‐visna virus strains was carried out with a panel of sheep antisera. Sheep antiserum to visna virus glycoprotein was the only one that did not show cross‐neutralization. The other sera showed varying degrees of cross‐reactivity.
H. J. Larsen, B. Hyllseth
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