Results 131 to 140 of about 316,007 (306)

Clinicopathological Characteristics and Survival Status of Male Breast Cancer: A Single Center Experience

open access: yesAsian Pacific Journal of Cancer Biology, 2017
Male breast cancer is a rare condition which causes to underlying mechanism is not well understood. Therefore, we have conducted this study to survey the clinicopathological characteristics of a series of male breast cancer consecutively treated in Yazd,
Seyed Mostafa Shiryazdi   +1 more
doaj  

Stage‐Specific H3K14 and H3K23 Succinylation Orchestrates Insect Metamorphosis and Oogenesis

open access: yesAdvanced Science, EarlyView.
Stage‐specific succinylation of histone H3 at lysine 14 and 23 differentially controls insect metamorphosis and oogenesis through distinct GPCR‐PKC signaling cascades, revealing an evolutionarily conserved epigenetic mechanism that coordinates key life‐history traits.
Yu‐Pu Jing   +9 more
wiley   +1 more source

The biology of male breast cancer

open access: yesThe Breast, 2018
Important differences have begun to emerge concerning the molecular profile of female and male breast cancer which may prove to be of therapeutic value. This review examined all the available data on the genomics of MBC. Most male cancers are ER+ve but without a corresponding increase in PR positivity and only a weaker association with estrogen ...
openaire   +2 more sources

Birth size and breast cancer risk: Re-analysis of individual participant data from 32 studied

open access: yes, 2008
Background Birth size, perhaps a proxy for prenatal environment, might be a correlate of subsequent breast cancer risk, but findings from epidemiological studies have been inconsistent.
Collaborative, GOP-NRFASROBC   +3 more
core  

Lactoferrin Deficiency During Lactation Causes Adult Obesity‐Related Metabolic Disease Through Persistent Adipose Dysfunction Driven by Impaired Adipocyte Development

open access: yesAdvanced Science, EarlyView.
Lactational lactoferrin deficiency exerts lasting effects on epididymal adipose tissue development from lactation into adulthood: it impairs adipocyte hyperplasia and induces pathological hypertrophy, resulting in lower body weight yet exacerbated metabolic dysfunction under a high‐fat diet in adulthood.
Qin An   +11 more
wiley   +1 more source

ZDHHC17‐Mediated CDK4 Palmitoylation Drives Cell Cycle Progression and Orchestrates Cancer Immune Surveillance

open access: yesAdvanced Science, EarlyView.
ZDHHC17 palmitoylates CDK4, which together with TRAF6‐mediated ubiquitination, drives cell cycle progression and immune surveillance, revealing a rational combination of CDK4 inhibitors with immune checkpoint blockers for ZDHHC17‐driven cancers. ABSTRACT Uncontrolled cell cycle progression is a hallmark of cancer, tightly regulated by both intrinsic ...
Zekang Wang   +7 more
wiley   +1 more source

Male breast cancer

open access: yesPlastic and Reconstructive Surgery, 1986
Z Y, Huang, H, Qian, J Y, Li
openaire   +2 more sources

Prognostic factors in node-negative male breast cancer

open access: yes, 2006
Male breast cancer has traditionally been compared with female breast cancer, using the same staging system and prognostic indicators. A variety of histochemical and pathologic factors commonly used in node-negative female breast cancer were applied to ...
Avisar, Eli   +3 more
core   +1 more source

Ferritinophagy Rewires Carnitine‐Dependent Lipid Metabolism to Inhibit PRRSV and IAV Replication

open access: yesAdvanced Science, EarlyView.
NCOA4‐mediated ferritinophagy reprograms carnitine metabolism by disrupting Fe‐S cluster biogenesis, thereby establishing an iron‐lipid axis that suppresses various viruses, including PRRSV and IAV. However, viruses counteract this mechanism by degrading NCOA4.
Kaifeng Guan   +7 more
wiley   +1 more source

Supplement use, vitamin C intake and breast cancer risk in UK women [PDF]

open access: yes, 2011
Background: When analysing relationships between breast cancer risk and vitamin C intake, few prospective studies have included vitamin C intake from supplements, or excluded general supplement users in dietary only analyses.
Hutchinson, Jayne
core  

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