Results 201 to 210 of about 1,087,549 (254)

Prednisolone Attenuates Improvement of Cardiac and Skeletal Contractile Function and Histopathology by Lisinopril and Spironolactone in the mdx Mouse Model of Duchenne Muscular Dystrophy

, 2014
Paul M.L. Janssen, Jason D. Murray, Kevin E. Schill, Neha Rastogi, Eric J. Schultz, Tam Tran, Subha V. Raman, Jill A. Rafael‐Fortney   +7 more
openalex   +2 more sources

Respiratory Control in the mdx Mouse Model of Duchenne Muscular Dystrophy

, 2015
David P. Burns, Deirdre Edge, Dervla O’Malley, Ken D. O’Halloran   +3 more
openalex   +2 more sources

Utrophin A is essential in mediating the functional adaptations of mdx mouse muscle following chronic AMPK activation.

Human Molecular Genetics, 2015
Hasanen Al-Rewashdy, Vladimir Ljubicic, Wei Lin, J. Renaud, B. Jasmin   +4 more
semanticscholar   +1 more source

Sunitinib promotes myogenic regeneration and mitigates disease progression in the mdx mouse model of Duchenne muscular dystrophy. [PDF]

Hum Mol Genet, 2019
Fontelonga TM, Jordan B, Nunes AM, Barraza-Flores P, Bolden N, Wuebbles RD, Griner LM, Hu X, Ferrer M, Marugan J, Southall N, Burkin DJ.   +11 more
europepmc   +1 more source

A two-site collaborative study of electrical impedance myography for evaluation of disease progression in murine Duchenne muscular dystrophy models. [PDF]

Sci Rep
Mantuano P, van Putten M, Cobb BS, Putker K, Boccanegra B, Tanganyika-de Winter C, Tulimiero L, Schneider AF, Cappellari O, Van De Vijver D, Engelbeen S, Pandeya S, Nagy JA, Rutkove S.   +13 more
europepmc   +1 more source

Some of the next articles are maybe not open access.

Related searches:

Dystrophin-negative slow-twitch soleus muscles are not susceptible to eccentric contraction induced injury over the lifespan of the mdx mouse.

American Journal of Physiology - Cell Physiology, 2021
Duchenne muscular dystrophy (DMD) is the second most common fatal genetic disease in humans and is characterized by the absence of a functional copy of the protein dystrophin from skeletal muscle.
L. Kiriaev, Sindy L. L Kueh, J. Morley, P. Houweling, Stephen Chan, K. North, S. Head   +6 more
semanticscholar   +1 more source

Mss51 deletion increases endurance and ameliorates histopathology in the mdx mouse model of Duchenne muscular dystrophy

The FASEB Journal, 2021
Mitochondrial derangement is an important contributor to the pathophysiology of muscular dystrophies and may be among the earliest cellular deficits. We have previously shown that disruption of Mss51, a mammalian skeletal muscle protein that localizes to
Yazmin I Rovira Gonzalez, Adam L. Moyer, Nicolas J LeTexier, August D Bratti, Siyuan Feng, Vanessa Peña, Congshan Sun, Hannah C. Pulcastro, Ting Liu, S. Iyer, R. Lovering, B. O’Rourke, K. Wagner   +12 more
semanticscholar   +1 more source

Muscular weakness in the mdx mouse

Journal of the Neurological Sciences, 1993
mdx mice are believed to be virtually free from neuromuscular symptoms, despite the presence of a degenerative/regenerative process that involves all skeletal muscles. We analyzed both the spontaneous motility and treadmill motor activity of mdx mice aged 15 days to 6 months.
Muntoni F., Mateddu A., Marchei F., Clerk A., SERRA, Gino   +4 more
openaire   +3 more sources

Neuromuscular transmission in the mdx mouse

Muscle & Nerve, 1990
AbstractThe mdx mouse is an animal model for human Duchenne dystrophy. In both disorders, the muscle fiber plasma membrane is rendered selectively vulnerable by dystrophin deficiency. In both disorders there are also ultrastructural abnormalities involving the postsynaptic membrane of the neuromuscular junction.
Alexandre Nagel, Andrew G. Engel, Frank Lehmann-Horn   +2 more
openaire   +3 more sources

Twitch and tetanus in mdx mouse muscle

Muscle & Nerve, 1992
AbstractWe compared mdx and C57BL10 anterior tibial muscle force in situ (single pulse, multiple pulse, staircase, posttetanic potentiation, and fatiguing stimulation patterns) to define muscle strength, physiology, and fatigue resistance. The relatively hypertrophied mdx muscle showed: reduced strength (N/cm2), an increased twitch‐tetanus ratio, and ...
John G. Quinlan, Sean P. Lyden, Scott R. Johnson, Mari K. McKee   +3 more
openaire   +3 more sources