Results 191 to 200 of about 1,251,059 (351)

Supplementum 283: Abstracts of the Swiss Oncology & Hematology Congress (SOHC)

open access: yesSwiss Medical Weekly
Swiss Society of Medical Oncology   +2 more
doaj   +1 more source

Targeting of PTP4A3 overexpression sensitises HGSOC cells towards chemotherapeutic drugs

open access: yesMolecular Oncology, EarlyView.
In HGSOC with normal KRAS expression, high PTP4A3 expression regulates autophagy activation. Conversely, in HGSOC with high KRAS expression, KRAS dictates autophagy control, and PTP4A3 is not required. When high PTP4A3 expression is inhibited, HGSOC cells are preferentially sensitised towards DNA‐damaging agents.
Ana López‐Garza   +3 more
wiley   +1 more source

Patterns and outcome of unplanned care in lung cancer patients: an observational study in a medical oncology department. [PDF]

open access: yesTransl Lung Cancer Res, 2023
Sánchez JC   +9 more
europepmc   +1 more source

Referral to Medical Oncology: A Crucial Step in the Treatment of Older Patients with Stage III Colon Cancer [PDF]

open access: bronze, 2006
RuiLi Luo   +4 more
openalex   +1 more source

Olaparib synergy screen reveals Exemestane induces replication stress in triple‐negative breast cancer

open access: yesMolecular Oncology, EarlyView.
Screening 166 FDA‐approved anticancer drugs identifies the aromatase inhibitor Exemestane as a synergistic partner of PARP inhibitor Olaparib in BRCA‐proficient triple‐negative breast cancer. Exemestane induces ROS‐mediated replication stress, enhancing DNA damage and apoptosis alongside Olaparib.
Nur Aininie Yusoh   +5 more
wiley   +1 more source

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