Results 31 to 40 of about 2,093 (92)

Immune‐Neuroendocrine Interactions and Autoimmune Diseases

open access: yesJournal of Immunology Research, Volume 13, Issue 2-4, Page 109-123, 2006., 2006
The relationship between immune‐neuroendocrine system is firmly established. The messengers of this connection are hormones, neuropeptides, neurotransmitters and cytokines. The immune‐neuroendocrine system have the capacity to synthesize and release these molecules, which, in turn, can stimulate or suppress the activity of immune or neuroendocrine ...
Luis J. Jara   +4 more
wiley   +1 more source

Self‐Reactivity and the Expression of Memory Markers Vary Independently in MRL‐Mp+/+ and MRL‐Mp‐lpr/lpr Mice

open access: yesJournal of Immunology Research, Volume 2, Issue 4, Page 309-318, 1992., 1992
MRL‐Mp‐lpr/lpr mice contain phenotypically abnormal populations of T cells, and exhibit an SLE‐like autoimmune disease in which autoantibodies are a prominent feature. We analyzed the phenotype and T‐cell receptor Vß expression pattern in CD4+ T cells of this mutant mouse strain to detect abnormalities that could explain the autoimmunity.
Lesley Smyth   +2 more
wiley   +1 more source

Inhibition of neutrophil infiltration and NETs formation ameliorates neuropsychiatric and renal dysfunction in MRL/lpr mice with lupus. [PDF]

open access: yesPLoS One
Deng Y   +15 more
europepmc   +1 more source

Cell-Targeted Inhibition of CaMK4 Suppresses Tertiary Lymphoid-like Structure Development in Lupus-Prone Mice. [PDF]

open access: yesInt J Mol Sci
Jamaly S   +7 more
europepmc   +1 more source

Mechanistic study of compound Muniziqi granule on lupus nephritis in MRL/lpr mice: insights from transcriptomics and experimental validation. [PDF]

open access: yesLupus Sci Med
Zhang J   +11 more
europepmc   +1 more source

IFNγ-associated immune-metabolic remodeling is linked to serotonin-kynurenine imbalance and cortical vulnerability in lupus-prone mice. [PDF]

open access: yesFront Immunol
Matmat K   +7 more
europepmc   +1 more source

Reduced late endosome/lysosome function promotes SLE through chronic PI3K activity and SHP-1/SHIP-1 defects. [PDF]

open access: yesJCI Insight
Kang S   +13 more
europepmc   +1 more source

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