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Macrophage migration inhibitory factor
Critical Care Medicine, 2005Macrophage migration inhibitory factor (MIF) has been proposed to be the physiologic counter-regulator of glucocorticoid action within the immune system. In this role, MIF's position within the cytokine cascade is to act in concert with glucocorticoids to control both the "set point" and the magnitude of the inflammatory response. As well as overriding
Lin, Leng, Richard, Bucala
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Macrophage migration inhibitory factor
Critical Care Medicine, 2002Macrophage migration inhibitory factor (MIF) has been proposed to be the physiologic counter-regulator of glucocorticoid action within the immune system. In this role, MIF's position within the cytokine cascade is to act in concert with glucocorticoids to control both the "set point" and the magnitude of the inflammatory response. As well as overriding
John A, Baugh, Richard, Bucala
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The Isolation of Migration Inhibition Factor
Immunological Communications, 1974Migration inhibition factor (MIF) has been separated from the supernates of tuberculin stimulated lymphocytes and from fetal calf serum by an affinity column of fucosamine bound to agarose beads. A glycoprotein with the characteristics of biologically active MIF and a molecular weight of approximately 50,000 Daltons was obtained by this one stage ...
R A, Fox, J M, MacSween
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Macrophage migration inhibitory factor
Expert Opinion on Therapeutic Targets, 2003Macrophage migration inhibitory factor (MIF) is a ubiquitous protein that is found in virtually all cells. Its precise function in the majority of cells is not known, but studies performed over the last decade indicate that it is a critical upstream regulator of the innate and acquired immune response.
Elias, Lolis, Richard, Bucala
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Cellular Immunology, 1981
Abstract Spleen and thymus cells from Swiss Webster ICR mice were separated into enriched subpopulations using anti-Lyt-1.2 and anti-Lyt-2.2, and tested for lymphokine production in response to Con A stimulation. The Lyt-1.2 subpopulation, helper cells, produces migration inhibition factor (MIF).
R A, Fox, K, Rajaraman
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Abstract Spleen and thymus cells from Swiss Webster ICR mice were separated into enriched subpopulations using anti-Lyt-1.2 and anti-Lyt-2.2, and tested for lymphokine production in response to Con A stimulation. The Lyt-1.2 subpopulation, helper cells, produces migration inhibition factor (MIF).
R A, Fox, K, Rajaraman
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Factor Mobility and Migration Models
2019The chapter introduces into the theory of factor mobility between regions or countries. After an introduction defining terms and motives for movements of factors in space it explains the basic factor mobility model assuming perfect competition and full factor price flexibility.
Bröcker, Johannes, Mitze, Timo Friedel
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