Results 211 to 220 of about 76,021 (334)
Mineralocorticoid Receptor-Dependent Adverse Remodeling After Myocardial Infarction Mediated by uNGALant Activation of NFκB [PDF]
Johann Bauersachs, Daniela Fraccarollo
openalex +1 more source
Abstract Background Albuminuria is associated with an increased risk of cardiovascular and kidney events. Sodium glucose co‐transporter 2 inhibitors (SGLT2i) reduce albuminuria and improve kidney outcomes in patients with albuminuric chronic kidney disease (CKD).
João Pedro Ferreira +10 more
wiley +1 more source
Licorice-Induced Pseudohyperaldosteronism Highlights an Underestimated Etiology of Hypertension. [PDF]
Bendjilali-Sabiani JJ +6 more
europepmc +1 more source
Sub-chronic stimulation of glucocorticoid receptor impairs and mineralocorticoid receptor protects cytosolic Ca2+ responses to glucose in pancreatic β-cells [PDF]
Masaru Koizumi, Toshihiko Yada
openalex +1 more source
Cortisol as mineralocorticoid [PDF]
openaire +2 more sources
Abstract Aim Glucagon‐like peptide 1 receptor agonists (GLP‐1RAs) have been established as effective treatments for type 2 diabetes, offering benefits beyond glycaemic control; however, their associations across multiple health outcomes remain insufficiently assessed.
Dongjin Yeo +8 more
wiley +1 more source
Dickkopf 3 in Urine: A Help for Prioritizing Management of Kidney Disease in Type 2 Diabetes. [PDF]
Rossing P.
europepmc +1 more source
Human mineralocorticoid receptor interacts with actin under mineralocorticoid ligand modulation [PDF]
Stéphan Jalaguier +4 more
openalex +1 more source
The biology of PKM2 in the metabolism and senescence in diabetic kidney disease
ABSTRACT Diabetic kidney disease (DKD) is one of the most common causes of chronic kidney disease that leads to end‐stage kidney disease, and its progression is closely linked to metabolic stress within renal tubular cells. Under long‐term hyperglycemia, cells shift their glucose metabolism from normal oxidative phosphorylation toward glycolysis.
Md. Imrul Kayes, Keizo Kanasaki
wiley +1 more source

