Results 221 to 230 of about 38,262 (274)

Controlled sustained delivery of monensin in cattle: The monensin R.D.D.®

Journal of Controlled Release, 1989
Abstract A delivery device (Monensin R.D.D.) providing continuous dosing of cattle with the growthpromoting agent monensin sodium was developed for use in range cattle. The device is capable of delivering approximately 100 mg monensin per day for up to 150 days. The device consists of a core composed of crystalline monensin sodium (40%) dispersed in
J.M. Conrad, D.S. Skinner
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Monensin in Broiler Rations

Avian Diseases, 1973
SUMMARY A total of 3750 broiler chicks were reared on the floor in two experiments replicated 8-12 times. Rate of growth, feed efficiency, mortality, and coccidial lesions were investigated comparing two anticoccidial agents and a negative control group. Lesion scores (severity of 1-4) were determined on sacrificed chicks at 3/2 and 7 weeks of age.
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Outbreak of monensin poisoning in cattle

Veterinary Record, 1983
Following accidental overdosage with monensin, nine deaths occurred in a batch of 40, four-to-seven-month-old calves. A description of the incident is given including the findings on post mortem examination. Inaccurate on-farm feed mixing can present problems as was highlighted by this outbreak.
D D, Wardrope, N S, Macleod, J R, Sloan
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Experimental bovine coccidiosis: Control with monensin

Veterinary Parasitology, 1986
Young Holstein-Friesian bull calves were used in a controlled experiment to evaluate the efficacy of monensin against coccidiosis. The calves were given oocysts of Eimeria bovis and/or E. zurnii. Medication was started 3 days prior to inoculation and continued during the 30-day experimental period. Oocyst shedding was quantified prior to and throughout
B E, Stromberg, J C, Schlotthauer, K J, Hamann, H, Saatara Oz, W J, Bemrick   +4 more
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Suspected monensin toxicosis in feedlot cattle

Australian Veterinary Journal, 1985
SummarySuspected monensin toxicosis was seen in feedlot cattle aged 6 to 9 months. Twenty cattle died following inclusion of monensin in the feed at 400g/tonne, which was 13 times the recommended level. The deaths occurred over 2 weeks. Clinical signs were inappetance, respiratory distress and sudden death.
R J, Geor, W F, Robinson
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Monensin Toxicity in Turkey Breeder Hens

Avian Diseases, 1989
High mortality in two flocks of 1900 turkey breeder hens accidentally fed 280 g monensin/ton of complete feed is described. Mortality attributed to the poisoning was 76% in flock 1 and 18% in flock 2. Clinically, turkeys were found dead, exhibited respiratory distress with wings extended laterally, had fine tremors, or showed posterior paresis and ...
M D, Ficken, D P, Wages, E, Gonder
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Bis-monensin: Synthesis of new chiral receptor from naturally occurring monensin ionophore

Tetrahedron Letters, 1990
Abstract Bis-monensin derivatives were synthesized and characterized for the first time as chiral receptor molecules. Among them, o -xylene-bridged bis-monensin exhibited particularly excellent chiral recognition ability by the inclusion of chiral ammonium guest cations in its central cavity.
Hiroshi Tsukube, Hajime Sohmiya
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Studies on Monensin Toxicity in Goats

Journal of Veterinary Medicine Series A, 1990
SummaryThis study was carried out to examine the effect of orally administered monensin on the liver of goats. Goats given monensin at the rate of 55 mg/kg of feed (55 ppm) for three weeks showed clinical signs of anorexia and diarrhea, along with increased pentobarbital sleeping time and serum SDH level indicating the possible presence of ...
R R, Dalvi, S G, Sawant
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Monensin toxicity in preruminant dairy heifers

Australian Veterinary Journal, 2003
Four‐to six‐week‐old calves from a seasonal dairy herd in North Western Tasmania were presented for veterinary attention due to the occurrence of sudden deaths. Necropsy examination of one of the calves revealed several small pale foci of 1cm diameter on the epicardium.
L J, Gabor, G M, Downing
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Effect of monensin on myoblast fusion

Biochemical and Biophysical Research Communications, 1985
Monensin, at a concentration of 0.5-10 microM, completely (100%) and reversibly inhibits fusion of embryonic chick myoblasts in vitro. At the same time, monensin administration leads to a marked accumulation of glycopeptides inside the cells and a decrease of those secreted into the medium.
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