Results 201 to 210 of about 9,063 (256)
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Current Protocols in Toxicology, 2000
AbstractMonoamine oxidase catalyzes the oxidative deamination of primary aliphatic and aromatic amines and some secondary and tertiary amines, including the hormone and neurotransmitter amines epinephrine, dopamine, norepinephrine and serotonin. The two isoenzymes, MAO‐A and MAO‐B, differ according to substrate and inhibitor specificities. For example,
Keith F, Tipton +2 more
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AbstractMonoamine oxidase catalyzes the oxidative deamination of primary aliphatic and aromatic amines and some secondary and tertiary amines, including the hormone and neurotransmitter amines epinephrine, dopamine, norepinephrine and serotonin. The two isoenzymes, MAO‐A and MAO‐B, differ according to substrate and inhibitor specificities. For example,
Keith F, Tipton +2 more
openaire +3 more sources
Acta Physiologica Scandinavica, 1972
AbstractHomogenates of the liver, the kidneys and the brain of the hedgehog, Erinaceus europaeus,. have been tested for monoamine oxidase activity. The enzyme was present in all 3 tissues, and the enzyme was found to act on all the substrates known to be oxidised by other mammalian sources of the enzyme.
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AbstractHomogenates of the liver, the kidneys and the brain of the hedgehog, Erinaceus europaeus,. have been tested for monoamine oxidase activity. The enzyme was present in all 3 tissues, and the enzyme was found to act on all the substrates known to be oxidised by other mammalian sources of the enzyme.
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Depression and monoamine oxidase
Progress in Neuro-Psychopharmacology, 19791. The first part of the paper is devoted to a critical review on the possible relationships between depression and monoamine oxidase. 2. This study describes the results of an investigation of MAO-activity in depression, using new approaches and methodology.
H, Coper +6 more
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Switching Monoamine Oxidase Inhibitors
Drug Intelligence & Clinical Pharmacy, 1985Substituting one monoamine oxidase inhibitor for another is recommended only after a drug-free interval to avoid hypertensive emergencies. The evidence and mechanism firmly supporting this caution is lacking. We report a case where monoamine oxidase inhibitors were substituted without apparent adverse consequences.
B L, True, B, Alexander, B, Carter
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Monoamine Oxidase Interactions
Journal of Clinical Psychopharmacology, 2023openaire +2 more sources
2015
Evidence for the genetic control of human MAO is now well established. The relationships of MAO activities to neuropsychiatric disorders or response to psychopharmacologic treatments are relatively unstudied. Preliminary findings from our own studies suggest that (1) blood MAO activity is a polygenically controlled trait; (2) there may be a subgroup of
D S, Robinson, A, Nies
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Evidence for the genetic control of human MAO is now well established. The relationships of MAO activities to neuropsychiatric disorders or response to psychopharmacologic treatments are relatively unstudied. Preliminary findings from our own studies suggest that (1) blood MAO activity is a polygenically controlled trait; (2) there may be a subgroup of
D S, Robinson, A, Nies
openaire +2 more sources
Integrative oncology: Addressing the global challenges of cancer prevention and treatment
Ca-A Cancer Journal for Clinicians, 2022Jun J Mao,, Msce +2 more
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