Results 141 to 150 of about 5,812 (165)
DNA Methylation in Gastric Cancer and Preneoplastic Lesions: Emerging Insights and Future Directions. [PDF]
Ceccon C +9 more
europepmc +1 more source
Prevalence of germline MSH3 polymorphisms in ulcerative colitis and early-onset colorectal cancer patients that potentiates inflammation-to-cancer transformation. [PDF]
Tseng-Rogenski SS +3 more
europepmc +1 more source
Enhanced transcriptomic profiling of esophageal tissue through optimized PAXgene fixation protocols. [PDF]
Charara F +9 more
europepmc +1 more source
Expression of human MutS homolog 2 (hMSH2) protein in resting and proliferating cells. [PDF]
The hMSH2 protein plays an important role in the DNA mismatch repair system. Since this system is involved in the correction of errors that occur during DNA replication, we studied the expression of hMSH2 protein in resting and DNA-replicating cells, as well as through the cell cycle in cell types with different growth characteristics.
Marra, Giancarlo +5 more
core +7 more sources
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Aquatic Toxicology, 2013
DNA mismatch repair (MMR) of simple base mismatches and small insertion-deletion loops in eukaryotes is initiated by the binding of the MutS homolog 2 (MSH2)-MSH6 heterodimer to mismatched DNA. Cadmium (Cd) is a genotoxic heavy metal that has been recognized as a human carcinogen.
Todd Hsu
exaly +3 more sources
DNA mismatch repair (MMR) of simple base mismatches and small insertion-deletion loops in eukaryotes is initiated by the binding of the MutS homolog 2 (MSH2)-MSH6 heterodimer to mismatched DNA. Cadmium (Cd) is a genotoxic heavy metal that has been recognized as a human carcinogen.
Todd Hsu
exaly +3 more sources
Regulation of human MutS homolog 2 (hMSH2) protein expression during the cell cycle. [PDF]
Marra G +6 more
openaire +2 more sources
Ai zheng = Aizheng = Chinese journal of cancer, 2004
Frequent loss of fragile histidine triad (FHIT) expression in human gastrointestinal tract carcinomas has been reported; however, there were divergent opinions regarding FHIT expression in colorectal carcinoma. Recent studies have suggested that FHIT inactivation can be a consequence of defects in mismatch repair proteins, particularly mut S homolog 2 (
Cheng-Cai, Yao +2 more
openaire +1 more source
Frequent loss of fragile histidine triad (FHIT) expression in human gastrointestinal tract carcinomas has been reported; however, there were divergent opinions regarding FHIT expression in colorectal carcinoma. Recent studies have suggested that FHIT inactivation can be a consequence of defects in mismatch repair proteins, particularly mut S homolog 2 (
Cheng-Cai, Yao +2 more
openaire +1 more source

