Results 201 to 210 of about 16,475 (253)

Energetic microdomains and the vascular control of neuronal and muscle excitability: Toward a unified model

The Journal of Physiology, EarlyView.
Abstract figure legend The capillary–mitochondria–ion channel (CMIC) axis scales structural resources to match functional workload. (Left) In settings of restricted energetic capacity (e.g. cortical neurons), sparse capillary networks and modest mitochondrial pools set a lower energetic ceiling, sufficient to support phasic, low‐workload excitability. (
L. Fernando Santana, Scott Earley
wiley   +1 more source

Myocardial Bridging Presenting With Ventricular Tachycardia and Non-ST-Segment Elevation Myocardial Infarction. [PDF]

JACC Case Rep
Devera JL, Amsterdam EA.
europepmc   +1 more source

Cellular mechanisms of radiation‐induced myocyte dysfunction: effects on calcium handling, ion channel regulation and mitochondrial energetics

The Journal of Physiology, EarlyView.
Abstract figure legend Dose‐dependent effects of radiation on cardiac electrophysiology and arrhythmia susceptibility. At low radiation doses (left), increased reactive oxygen species (ROS) initiate an ROS–Ca2+ positive feedback loop involving calcium/calmodulin‐dependent protein kinase II (CaMKII) activation, enhanced L‐type Ca2+ current (ICaL ...
Hannah M. Zukowski, Colleen E. Clancy
wiley   +1 more source

Multiple Bands of Myocardial Bridging Involving the Left Anterior Descending and Posterior Descending Arteries: A Case Report and Its Clinical Implications. [PDF]

Cureus
Abouzaid KA, Imam A, Basyouny A, Ooi V, Coffin MK.   +4 more
europepmc   +1 more source

The importance of cross‐talk in research and the body: An early career researcher perspective

Experimental Physiology, EarlyView.
Lucy J. Ryan, Jamie A. Elliott, Robert Little, Mhairi A. Paul   +3 more
wiley   +1 more source

Small‐conductance Ca2⁺‐activated K⁺ channels in cardiac excitation–contraction coupling: Bridging mitochondria, sarcolemma and antiarrhythmic therapy

The Journal of Physiology, EarlyView.
Abstract figure legend Mitochondrial SK channel enhancement reduces cardiac arrhythmia trigger. Spontaneous sarcoplasmic reticulum (SR) Ca2+ release via hyperactive RyR2s underlies an increased arrhythmia trigger, promoting early and delayed afterdepolarizations during stress. Hyperactive RyR2s causes rise in cytosolic [Ca2+] during diastole. Clearance
Dmitry Terentyev, Alexey V. Glukhov, Vladimir Bogdanov, Aleksei S. Averin, Roland Veress, Bum‐Rak Choi, Shanna Hamilton, Andriy E. Belevych   +7 more
wiley   +1 more source

Myocardial bridging resulting in an atypical acute coronary syndrome in a young athlete: a case report. [PDF]

Eur Heart J Case Rep
Coulibaly S, Genet T, Bisson A, Bernard A, Ivanes F.   +4 more
europepmc   +1 more source

Caenorhabditis elegans as an in vivo model system for human inherited primary arrhythmia syndromes

The Journal of Physiology, EarlyView.
Abstract figure legend Most genes involved in inherited primary arrhythmia syndromes (IPAS) are conserved in Caenorhabditis elegans, where genetic manipulation enables functional characterization of variants, identification of regulatory proteins, and in vivo drug testing.
Antoine Delinière, Thomas Boulin, Maëlle Jospin, Alexandre Janin, Gilles Millat, Philippe Chevalier, Olga Andrini   +6 more
wiley   +1 more source

Coronary vasospasm and cardiovascular outcomes in patients with isolated myocardial bridging: A retrospective study. [PDF]

Cardiol J
Heo Y, Oh S, Cho KH, Kim MC, Sim DS, Hong YJ, Kim JH, Ahn Y, Jeong MH.   +8 more
europepmc   +1 more source

Extensive Myocardial Bridging Treated with CABG. [PDF]

J Brown Hosp Med
Fan J, Sanford C, Zamin S, Jones BD.
europepmc   +1 more source