Results 121 to 130 of about 19,191,137 (252)

Philosophy Born of Struggle 2007: Africa in the Dialectics of History

open access: yes, 2015
Panel 1 with presenter Albert ...
Philosophy Born of Struggle Conference
core  

Regulation of the lncRNA NEAT1 by p53‐ΔNp63 crosstalk modulates the DNA damage response and therapeutic efficacy in HNSCC

open access: yesMolecular Oncology, EarlyView.
In head and neck squamous cell carcinoma (HNSCC) p53 and p63 exert opposite roles on the transcription regulation of the lncRNA NEAT1. Under basal conditions, p53 levels are low and p63 represses NEAT1 expression. Upon genotoxic stress, p53 is rapidly induced, displacing p63 from the NEAT1 promoter leading to NEAT1 transcriptional activation and ...
Sara De Domenico   +5 more
wiley   +1 more source

The Rev James Lawson: The Non-Violence Struggle 10-16-2007 1 of 2

open access: yes, 2007
Includes descriptive metadata provided by producer in MP4 file: "The Non-Violence Struggle - Video - The Rev James Lawson The Non-Violence Struggle 10-16-2007 1 of 2." Videorecording of a class session.
Lawson, James M., 1928-
core  

APOBEC3 activity and DNA polymerase‐ε deficiency are associated with distinct IDH1 R132 hotspot mutations

open access: yesMolecular Oncology, EarlyView.
Isocitrate dehydrogenase 1 (IDH1) mutations are highly recurrent in multiple human cancer types, including cholangiocarcinoma and glioma. IDH1 R132C is the most common IDH1 mutation in cholangiocarcinoma and likely arises from APOBEC3A‐ or APOBEC3B‐mediated deamination.
Kelly E. Butler   +3 more
wiley   +1 more source

Areas administered by the National Park Service /

open access: yes, 1949
Vol. for 1965 includes the Registry of national landmarks.None published for 1958.Vol. for 1965 includes the Registry of national landmarks.Mode of access: Internet.Continued by: National parks & landmarks.Continues: National park system and other areas ...
United States. National Park Service.
core  

p190A/ARHGAP35 and p190B/ARHGAP5 proteins in endometrial cancer: a novel cancer‐relevant paralog interplay

open access: yesMolecular Oncology, EarlyView.
This study identifies ARHGAP5, in addition to the frequently mutated ARHGAP35, as significantly mutated in endometrial cancer. Mutations in both genes co‐occur and are associated with their correlated downregulation. Functional CRISPR studies show that both paralogs regulate similar pathways, including actin cytoskeleton organization.
Mathilde Pinault   +12 more
wiley   +1 more source

The Rev James Lawson: The Non-Violence Struggle 10-16-2007 2 of 2

open access: yes, 2007
Includes descriptive metadata provided by producer in MP4 file: "The Non-Violence Struggle - Video - The Rev James Lawson The Non-Violence Struggle 10-16-2007 2 of 2." Videorecording of a class session.
Lawson, James M., 1928-
core  

Extracellular matrix remodeling and immune reprogramming drive residual tumor progression of liver cancer after incomplete microwave ablation

open access: yesMolecular Oncology, EarlyView.
Incomplete microwave ablation (iMWA) of liver cancer triggers a biphasic progression in residual tumors. At Day 3, the microenvironment is characterized by acute inflammatory responses and extracellular matrix (ECM) remodeling. By Day 14, a profound shift occurs toward oncogenic signal transduction and immunosuppression, marked by macrophage ...
Yu Liu   +9 more
wiley   +1 more source

National Security Strategy of the Republic of Poland, 2003

open access: yes, 2003
"Ongoing changes in the international security environment are reflected in this document that introduces a reassessment and adjustment of Poland’s national security strategy.
Ministry of National Defence of Poland
core  

SPHINX31 acts as a SRPK1 inhibitor targeting the ATR/DNA‐PKcs/CHK1 replicative checkpoint to inhibit cell growth in non‐small cell lung cancer

open access: yesMolecular Oncology, EarlyView.
The kinase SRPK1 directly interacts with the protein TOPBP1 and regulates the pre‐mRNA splicing of WIZ thereby contributing to the activation of the ATR/CHK1 replicative checkpoint in response to replicative stress. This allows cancer cells' genomic stability and survival.
Amani Shreim   +17 more
wiley   +1 more source

Home - About - Disclaimer - Privacy